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10.1016/j.dib.2016.03.105 http://scihub22266oqcxt.onion/10.1016/j.dib.2016.03.105 C4865660!4865660!27222853     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Data+Brief 2016 ; 7 (ä): 1560-4 Nephropedia Template TP {{tp|p=27222853|t=2016. Data on sulforaphane treatment mediated suppression of autoreactive, inflammatory M1 macrophages |pdf=|usr=}}{{27222853}} gab.com Text Data on sulforaphane treatment mediated suppression of autoreactive, inflammatory M1 macrophages JO: Data Brief http://www.kidney.de/pget.php?&tw=1&pmid=27222853 #FOAvip Any chronic, inflammatory, autoimmune disease (e.g. arthritis) associated pathogenesis directs uncontrolled accumulation of both soluble forms of collagens in the synovial fluids and M1 macrophages around inflamed tissues. Despite of few studies demonstrating efficiency of Sulforaphane (SFN) in suppressing arthritis associated collagen restricted T cells or fibroblasts, its effects on macrophage polarity and plasticity are less understood. Recently, we reported regulation of phenotypic and functional switching by SFN in induced and spontaneously differentiating human monocytes [1]. Here, flow cytometry, western blot and ELISA derived data demonstrated that SFN inhibited in vitro inflammatory responses developed by soluble human collagens (I?IV) induced auto-reactive M1 type monocyte/macrophage model. Twit Text FOAVip Data on sulforaphane treatment mediated suppression of autoreactive, inflammatory M1 macrophages ????Data Brief http://www.kidney.de/pget.php?&tw=1&pmid=27222853 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27222853 #FOAvip English Wikipedia <ref>{{Cite pmid|27222853}}</ref> Data on sulforaphane treatment mediated suppression of autoreactive, inflammatory M1 macrophages #MMPMID27222853 Pal S; Konkimalla VB Data Brief 2016[Jun]; 7 (ä): 1560-4 PMID27222853show ga Any chronic, inflammatory, autoimmune disease (e.g. arthritis) associated pathogenesis directs uncontrolled accumulation of both soluble forms of collagens in the synovial fluids and M1 macrophages around inflamed tissues. Despite of few studies demonstrating efficiency of Sulforaphane (SFN) in suppressing arthritis associated collagen restricted T cells or fibroblasts, its effects on macrophage polarity and plasticity are less understood. Recently, we reported regulation of phenotypic and functional switching by SFN in induced and spontaneously differentiating human monocytes [1]. Here, flow cytometry, western blot and ELISA derived data demonstrated that SFN inhibited in vitro inflammatory responses developed by soluble human collagens (I?IV) induced auto-reactive M1 type monocyte/macrophage model. äData on sulforaphane treatment mediated suppression of autoreactive, i(epmc).pdf DeepDyve Pubget Overpricing