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10.1016/j.yjmcc.2016.03.005 http://scihub22266oqcxt.onion/10.1016/j.yjmcc.2016.03.005 C4861678!4861678 !26996756     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26996756 &cmd=llinks): Failed to open stream: HTTP request failed! 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Myocyte-fibroblast communication in cardiac fibrosis and arrhythmias: Mechanisms and model systems |pdf=|usr=}}{{26996756 }} gab.com Text Myocyte-fibroblast communication in cardiac fibrosis and arrhythmias: Mechanisms and model systems JO: J Mol Cell Cardiol http://www.kidney.de/pget.php?&tw=1&pmid=26996756 #FOAvip Development of cardiac fibrosis and arrhythmias is controlled by the activity of and communication between cardiomyocytes and fibroblasts in the heart. Myocyte-fibroblast interactions occur via both direct and indirect means including paracrine mediators, extracellular matrix interactions, electrical modulators, mechanical junctions, and membrane nanotubes. In the diseased heart, cardiomyocyte and fibroblast ratios and activity, and thus myocyte-fibroblast interactions, change and are thought to contribute to the course of disease including development of fibrosis and arrhythmogenic activity. Fibroblasts have a developing role in modulating cardiomyocyte electrical and hypertrophic activity, however gaps in knowledge regarding these interactions still exist. Research in this field has necessitated the development of unique approaches to isolate and control myocyte-fibroblast interactions. Numerous methods for 2D and 3D co-culture systems have been developed, while a growing part of this field is in the use of better tools for in vivo systems including cardiomyocyte and fibroblast specific Cre mouse lines for cell type specific genetic ablation. This review will focus on (i) mechanisms of myocyte-fibroblast communication and their effects on disease features such as cardiac fibrosis and arrhythmias as well as (ii) methods being used and currently developed in this field. Twit Text FOAVip Myocyte-fibroblast communication in cardiac fibrosis and arrhythmias: Mechanisms and model systems ????J Mol Cell Cardiol http://www.kidney.de/pget.php?&tw=1&pmid=26996756 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26996756 #FOAvip English Wikipedia <ref>{{Cite pmid|26996756 }}</ref> Myocyte-fibroblast communication in cardiac fibrosis and arrhythmias: Mechanisms and model systems #MMPMID26996756 Pellman J ; Zhang J ; Sheikh F J Mol Cell Cardiol 2016[May]; 94 (ä): 22-31 PMID26996756 show ga Development of cardiac fibrosis and arrhythmias is controlled by the activity of and communication between cardiomyocytes and fibroblasts in the heart. Myocyte-fibroblast interactions occur via both direct and indirect means including paracrine mediators, extracellular matrix interactions, electrical modulators, mechanical junctions, and membrane nanotubes. In the diseased heart, cardiomyocyte and fibroblast ratios and activity, and thus myocyte-fibroblast interactions, change and are thought to contribute to the course of disease including development of fibrosis and arrhythmogenic activity. Fibroblasts have a developing role in modulating cardiomyocyte electrical and hypertrophic activity, however gaps in knowledge regarding these interactions still exist. Research in this field has necessitated the development of unique approaches to isolate and control myocyte-fibroblast interactions. Numerous methods for 2D and 3D co-culture systems have been developed, while a growing part of this field is in the use of better tools for in vivo systems including cardiomyocyte and fibroblast specific Cre mouse lines for cell type specific genetic ablation. This review will focus on (i) mechanisms of myocyte-fibroblast communication and their effects on disease features such as cardiac fibrosis and arrhythmias as well as (ii) methods being used and currently developed in this field. |*Cell Communication [MESH] |Animals [MESH] |Arrhythmias, Cardiac/*metabolism/physiopathology [MESH] |Coculture Techniques [MESH] |Disease Models, Animal [MESH] |Extracellular Matrix Proteins/metabolism [MESH] |Extracellular Matrix/metabolism [MESH] |Fibroblasts/*metabolism [MESH] |Fibrosis [MESH] |Humans [MESH] |In Vitro Techniques [MESH] |Models, Biological [MESH] |Myocardium/*metabolism/pathology [MESH]Myocyte-fibroblast communication in cardiac fibrosis and arrhythmias: (epmc).pdf DeepDyve Pubget Overpricing