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10.1016/j.cell.2011.05.019 http://scihub22266oqcxt.onion/10.1016/j.cell.2011.05.019 C4858411!4858411!21663790     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=21663790&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Cell 2011 ; 145 (6): 835-50 Nephropedia Template TP {{tp|p=21663790|t=2011. Chromatin connections to pluripotency and cellular reprogramming |pdf=|usr=}}{{21663790}} gab.com Text Chromatin connections to pluripotency and cellular reprogramming JO: Cell http://www.kidney.de/pget.php?&tw=1&pmid=21663790 #FOAvip The pluripotent state of embryonic stem (ES) cells provides a unique perspective on regulatory programs that govern self-renewal and differentiation, and somatic cell reprogramming. Here we review the highly connected protein and transcriptional networks that maintain pluripotency, and how they are intertwined with factors that affect chromatin structure and function. The complex interrelationships between pluripotency and chromatin factors are illustrated by X-chromosome inactivation, regulatory control by non-coding RNAs, and environmental influences on cell states. Recent findings suggest a model in which environmental cues and growth conditions may direct the fate of cells transitioning a ?plastic state? induced during reprogramming. Twit Text FOAVip Chromatin connections to pluripotency and cellular reprogramming ????Cell http://www.kidney.de/pget.php?&tw=1&pmid=21663790 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=21663790 #FOAvip English Wikipedia <ref>{{Cite pmid|21663790}}</ref> Chromatin connections to pluripotency and cellular reprogramming #MMPMID21663790 Orkin SH; Hochedlinger K Cell 2011[Jun]; 145 (6): 835-50 PMID21663790show ga The pluripotent state of embryonic stem (ES) cells provides a unique perspective on regulatory programs that govern self-renewal and differentiation, and somatic cell reprogramming. Here we review the highly connected protein and transcriptional networks that maintain pluripotency, and how they are intertwined with factors that affect chromatin structure and function. The complex interrelationships between pluripotency and chromatin factors are illustrated by X-chromosome inactivation, regulatory control by non-coding RNAs, and environmental influences on cell states. Recent findings suggest a model in which environmental cues and growth conditions may direct the fate of cells transitioning a ?plastic state? induced during reprogramming. äChromatin connections to pluripotency and cellular reprogramming (epmc).pdf DeepDyve Pubget Overpricing