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10.1016/j.bbrc.2015.11.060 http://scihub22266oqcxt.onion/10.1016/j.bbrc.2015.11.060 C4854783!4854783 !26626076     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 245.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\26626076 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Biochem+Biophys+Res+Commun 2016 ; 473 (3 ): 698-703 Nephropedia Template TP {{tp|p=26626076 |t=2016. The role of Wnt regulation in heart development, cardiac repair and disease: A tissue engineering perspective |pdf=|usr=}}{{26626076 }} gab.com Text The role of Wnt regulation in heart development, cardiac repair and disease: A tissue engineering perspective JO: Biochem Biophys Res Commun http://www.kidney.de/pget.php?&tw=1&pmid=26626076 #FOAvip Wingless-related integration site (Wnt) signaling has proven to be a fundamental mechanism in cardiovascular development as well as disease. Understanding its particular role in heart formation has helped to develop pluripotent stem cell differentiation protocols that produce relatively pure cardiomyocyte populations. The resultant cardiomyocytes have been used to generate heart tissue for pharmaceutical testing, and to study physiological and disease states. Such protocols in combination with induced pluripotent stem cell technology have yielded patient-derived cardiomyocytes that exhibit some of the hallmarks of cardiovascular disease and are therefore being used to model disease states. While FDA approval of new treatments typically requires animal experiments, the burgeoning field of tissue engineering could act as a replacement. This would necessitate the generation of reproducible three-dimensional cardiac tissues in a well-controlled environment, which exhibit native heart properties, such as cellular density, composition, extracellular matrix composition, and structure-function. Such tissues could also enable the further study of Wnt signaling. Furthermore, as Wnt signaling has been found to have a mechanistic role in cardiac pathophysiology, e.g. heart attack, hypertrophy, atherosclerosis, and aortic stenosis, its strategic manipulation could provide a means of generating reproducible and specific, physiological and pathological cardiac models. Twit Text FOAVip The role of Wnt regulation in heart development, cardiac repair and disease: A tissue engineering perspective ????Biochem Biophys Res Commun http://www.kidney.de/pget.php?&tw=1&pmid=26626076 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26626076 #FOAvip English Wikipedia <ref>{{Cite pmid|26626076 }}</ref> The role of Wnt regulation in heart development, cardiac repair and disease: A tissue engineering perspective #MMPMID26626076 Pahnke A ; Conant G ; Huyer LD ; Zhao Y ; Feric N ; Radisic M Biochem Biophys Res Commun 2016[May]; 473 (3 ): 698-703 PMID26626076 show ga Wingless-related integration site (Wnt) signaling has proven to be a fundamental mechanism in cardiovascular development as well as disease. Understanding its particular role in heart formation has helped to develop pluripotent stem cell differentiation protocols that produce relatively pure cardiomyocyte populations. The resultant cardiomyocytes have been used to generate heart tissue for pharmaceutical testing, and to study physiological and disease states. Such protocols in combination with induced pluripotent stem cell technology have yielded patient-derived cardiomyocytes that exhibit some of the hallmarks of cardiovascular disease and are therefore being used to model disease states. While FDA approval of new treatments typically requires animal experiments, the burgeoning field of tissue engineering could act as a replacement. This would necessitate the generation of reproducible three-dimensional cardiac tissues in a well-controlled environment, which exhibit native heart properties, such as cellular density, composition, extracellular matrix composition, and structure-function. Such tissues could also enable the further study of Wnt signaling. Furthermore, as Wnt signaling has been found to have a mechanistic role in cardiac pathophysiology, e.g. heart attack, hypertrophy, atherosclerosis, and aortic stenosis, its strategic manipulation could provide a means of generating reproducible and specific, physiological and pathological cardiac models. |*Gene Expression Regulation [MESH] |Animals [MESH] |Aortic Valve Stenosis/physiopathology [MESH] |Cell Differentiation [MESH] |Heart Diseases/*therapy [MESH] |Heart/physiology/*physiopathology [MESH] |Humans [MESH] |Myocardium/*metabolism [MESH] |Signal Transduction [MESH] |Swine [MESH] |Tissue Engineering/*methods [MESH] |Wnt Proteins/*metabolism [MESH]The role of Wnt regulation in heart development, cardiac repair and di(epmc).pdf DeepDyve Pubget Overpricing