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10.1016/j.yjmcc.2015.11.016 http://scihub22266oqcxt.onion/10.1016/j.yjmcc.2015.11.016 C4846511!4846511!26593723     free     free     free       J+Mol+Cell+Cardiol 2016 ; 93 (ä): 143-8 Nephropedia Template TP {{tp|p=26593723|t=2016. Role of Inflammatory Cells in Fibroblast Activation |pdf=|usr=}}{{26593723}} gab.com Text Role of Inflammatory Cells in Fibroblast Activation JO: J Mol Cell Cardiol http://www.kidney.de/pget.php?&tw=1&pmid=26593723 #FOAvip Although fibrosis is an essential response to acute cardiac tissue injury, prolonged myofibroblast activation and progressive fibrosis lead to further distortion of tissue architecture and worsened cardiac function. Thus, optimal tissue repair following injury requires tight control over myofibroblast activation. It is now recognized that inflammation plays a critical role in regulating fibrosis. In this review we will highlight how advances in the field of innate immunity have led to a better understanding of the role of inflammation in cardiovascular disease and, in particular, in the regulation of fibrosis. Specifically, we will discuss how the innate immune system recognizes tissue damage in settings of acute injury and chronic cardiovascular disease. We will also review the role of different cell populations in this response, particularly the unique role of different macrophage subsets and mast cells. Twit Text FOAVip Role of Inflammatory Cells in Fibroblast Activation ????J Mol Cell Cardiol http://www.kidney.de/pget.php?&tw=1&pmid=26593723 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26593723 #FOAvip English Wikipedia <ref>{{Cite pmid|26593723}}</ref> Role of Inflammatory Cells in Fibroblast Activation #MMPMID26593723 Hartupee J; Mann DL J Mol Cell Cardiol 2016[Apr]; 93 (ä): 143-8 PMID26593723show ga Although fibrosis is an essential response to acute cardiac tissue injury, prolonged myofibroblast activation and progressive fibrosis lead to further distortion of tissue architecture and worsened cardiac function. Thus, optimal tissue repair following injury requires tight control over myofibroblast activation. It is now recognized that inflammation plays a critical role in regulating fibrosis. In this review we will highlight how advances in the field of innate immunity have led to a better understanding of the role of inflammation in cardiovascular disease and, in particular, in the regulation of fibrosis. Specifically, we will discuss how the innate immune system recognizes tissue damage in settings of acute injury and chronic cardiovascular disease. We will also review the role of different cell populations in this response, particularly the unique role of different macrophage subsets and mast cells. äRole of Inflammatory Cells in Fibroblast Activation (epmc).pdf DeepDyve Pubget Overpricing