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10.1111/ajt.13660 http://scihub22266oqcxt.onion/10.1111/ajt.13660 C4844852!4844852!26696401     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Am+J+Transplant 2016 ; 16 (5): 1358-64 Nephropedia Template TP {{tp|p=26696401|t=2016. Environmental Exposures ? The Missing Link in Immune Responses After Transplantation |pdf=|usr=}}{{26696401}} gab.com Text Environmental Exposures The Missing Link in Immune Responses After Transplantation JO: Am J Transplant http://www.kidney.de/pget.php?&tw=1&pmid=26696401 #FOAvip In transplantation, immunosuppression has been directed at controlling acute responses, but treatment of chronic rejection has been ineffective. It is possible that factors that have previously been unaccounted for, such as exposure to inhaled pollution, ultraviolet light, or loss of the normal equilibrium between the gut immune system and the outside environment may be responsible for shifting immune responses to an effector/inflammatory phenotype, which leads to loss of self-tolerance and graft acceptance, and a shift towards autoimmunity and chronic rejection. Cells of the immune system are in a constant balance of effector response, regulation, and quiescence. Endogenous and exogenous signals can shift this balance through the aryl hydrocarbon receptor, which serves as a thermostat to modulate the response one way or the other, both at mucosal surfaces of interface organs to the outside environment, and in the internal milieu. Better understanding of this balance will identify a target for maintenance of self-tolerance and continued graft acceptance in patients who have achieved a ?steady state? after transplantation. Twit Text FOAVip Environmental Exposures The Missing Link in Immune Responses After Transplantation ????Am J Transplant http://www.kidney.de/pget.php?&tw=1&pmid=26696401 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26696401 #FOAvip English Wikipedia <ref>{{Cite pmid|26696401}}</ref> Environmental Exposures ? The Missing Link in Immune Responses After Transplantation #MMPMID26696401 Julliard W; Owens L; O?Driscoll C; Fechner J; Mezrich J Am J Transplant 2016[May]; 16 (5): 1358-64 PMID26696401show ga In transplantation, immunosuppression has been directed at controlling acute responses, but treatment of chronic rejection has been ineffective. It is possible that factors that have previously been unaccounted for, such as exposure to inhaled pollution, ultraviolet light, or loss of the normal equilibrium between the gut immune system and the outside environment may be responsible for shifting immune responses to an effector/inflammatory phenotype, which leads to loss of self-tolerance and graft acceptance, and a shift towards autoimmunity and chronic rejection. Cells of the immune system are in a constant balance of effector response, regulation, and quiescence. Endogenous and exogenous signals can shift this balance through the aryl hydrocarbon receptor, which serves as a thermostat to modulate the response one way or the other, both at mucosal surfaces of interface organs to the outside environment, and in the internal milieu. Better understanding of this balance will identify a target for maintenance of self-tolerance and continued graft acceptance in patients who have achieved a ?steady state? after transplantation. äEnvironmental Exposures ? The Missing Link in Immune Responses After T(epmc).pdf DeepDyve Pubget Overpricing