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10.1016/j.cell.2016.02.060 http://scihub22266oqcxt.onion/10.1016/j.cell.2016.02.060 C4826480!4826480!27058667     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Cell 2016 ; 165 (2): 475-87 Nephropedia Template TP {{tp|p=27058667|t=2016. The CDK-APC/C Oscillator Predominantly Entrains Periodic Cell-Cycle Transcription |pdf=|usr=}}{{27058667}} gab.com Text The CDK-APC/C Oscillator Predominantly Entrains Periodic Cell-Cycle Transcription JO: Cell http://www.kidney.de/pget.php?&tw=1&pmid=27058667 #FOAvip Throughout cell cycle progression, the expression of multiple transcripts oscillate, and whether these are under the centralized control of the CDK-APC/C proteins or can be driven by a de-centralized transcription factor (TF) cascade is a fundamental question for understanding cell cycle regulation. In budding yeast, we find that the transcription of nearly all genes, as assessed by RNA-seq or fluorescence microscopy in single cells, is dictated by CDK-APC/C. Three exceptional genes are transcribed in a pulsatile pattern in a variety of CDK-APC/C arrests. Pursuing one of these transcripts, the SIC1 inhibitor of B-type cyclins, we use a combination of mathematical modeling and experimentation to provide evidence that, counter-intuitively, Sic1 provides a failsafe mechanism promoting nuclear division when levels of mitotic cyclins are low. Twit Text FOAVip The CDK-APC/C Oscillator Predominantly Entrains Periodic Cell-Cycle Transcription ????Cell http://www.kidney.de/pget.php?&tw=1&pmid=27058667 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27058667 #FOAvip English Wikipedia <ref>{{Cite pmid|27058667}}</ref> The CDK-APC/C Oscillator Predominantly Entrains Periodic Cell-Cycle Transcription #MMPMID27058667 Rahi SJ; Pecani K; Ondracka A; Oikonomou C; Cross FR Cell 2016[Apr]; 165 (2): 475-87 PMID27058667show ga Throughout cell cycle progression, the expression of multiple transcripts oscillate, and whether these are under the centralized control of the CDK-APC/C proteins or can be driven by a de-centralized transcription factor (TF) cascade is a fundamental question for understanding cell cycle regulation. In budding yeast, we find that the transcription of nearly all genes, as assessed by RNA-seq or fluorescence microscopy in single cells, is dictated by CDK-APC/C. Three exceptional genes are transcribed in a pulsatile pattern in a variety of CDK-APC/C arrests. Pursuing one of these transcripts, the SIC1 inhibitor of B-type cyclins, we use a combination of mathematical modeling and experimentation to provide evidence that, counter-intuitively, Sic1 provides a failsafe mechanism promoting nuclear division when levels of mitotic cyclins are low. äThe CDK-APC/C Oscillator Predominantly Entrains Periodic Cell-Cycle Tr(epmc).pdf DeepDyve Pubget Overpricing