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10.1165/rcmb.2015-0323TR http://scihub22266oqcxt.onion/10.1165/rcmb.2015-0323TR C4821060!4821060!26744837     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Am+J+Respir+Cell+Mol+Biol 2016 ; 54 (4): 451-60 Nephropedia Template TP {{tp|p=26744837|t=2016. Endothelial and Smooth Muscle Cell Interactions in the Pathobiology of Pulmonary Hypertension |pdf=|usr=}}{{26744837}} gab.com Text Endothelial and Smooth Muscle Cell Interactions in the Pathobiology of Pulmonary Hypertension JO: Am J Respir Cell Mol Biol http://www.kidney.de/pget.php?&tw=1&pmid=26744837 #FOAvip In the pulmonary vasculature, the endothelial and smooth muscle cells are two key cell types that play a major role in the pathobiology of pulmonary vascular disease and pulmonary hypertension. The normal interactions between these two cell types are important for the homeostasis of the pulmonary circulation, and any aberrant interaction between them may lead to various disease states including pulmonary vascular remodeling and pulmonary hypertension. It is well recognized that the endothelial cell can regulate the function of the underlying smooth muscle cell by releasing various bioactive agents such as nitric oxide and endothelin-1. In addition to such paracrine regulation, other mechanisms exist by which there is cross-talk between these two cell types, including communication via the myoendothelial injunctions and information transfer via extracellular vesicles. Emerging evidence suggests that these nonparacrine mechanisms play an important role in the regulation of pulmonary vascular tone and the determination of cell phenotype and that they are critically involved in the pathobiology of pulmonary hypertension. Twit Text FOAVip Endothelial and Smooth Muscle Cell Interactions in the Pathobiology of Pulmonary Hypertension ????Am J Respir Cell Mol Biol http://www.kidney.de/pget.php?&tw=1&pmid=26744837 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26744837 #FOAvip English Wikipedia <ref>{{Cite pmid|26744837}}</ref> Endothelial and Smooth Muscle Cell Interactions in the Pathobiology of Pulmonary Hypertension #MMPMID26744837 Gao Y; Chen T; Raj JU Am J Respir Cell Mol Biol 2016[Apr]; 54 (4): 451-60 PMID26744837show ga In the pulmonary vasculature, the endothelial and smooth muscle cells are two key cell types that play a major role in the pathobiology of pulmonary vascular disease and pulmonary hypertension. The normal interactions between these two cell types are important for the homeostasis of the pulmonary circulation, and any aberrant interaction between them may lead to various disease states including pulmonary vascular remodeling and pulmonary hypertension. It is well recognized that the endothelial cell can regulate the function of the underlying smooth muscle cell by releasing various bioactive agents such as nitric oxide and endothelin-1. In addition to such paracrine regulation, other mechanisms exist by which there is cross-talk between these two cell types, including communication via the myoendothelial injunctions and information transfer via extracellular vesicles. Emerging evidence suggests that these nonparacrine mechanisms play an important role in the regulation of pulmonary vascular tone and the determination of cell phenotype and that they are critically involved in the pathobiology of pulmonary hypertension. äEndothelial and Smooth Muscle Cell Interactions in the Pathobiology of(epmc).pdf DeepDyve Pubget Overpricing