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2016 ; 2016
(ä): 3240131
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A Case of Primary Hypoparathyroidism Presenting with Acute Kidney Injury
Secondary to Rhabdomyolysis
#MMPMID27034860
Sumnu A
; Aydin Z
; Gursu M
; Uzun S
; Karadag S
; Cebeci E
; Ozturk S
; Kazancioglu R
Case Rep Nephrol
2016[]; 2016
(ä): 3240131
PMID27034860
show ga
Hypoparathyroidism is the most common cause of symmetric calcification of the
basal ganglia. Herein, a case of primary hypoparathyroidism with severe tetany,
rhabdomyolysis, and acute kidney injury is presented. A 26-year-old male was
admitted to the emergency clinic with leg pain and cramps, nausea, vomiting, and
decreased amount of urine. He had been treated for epilepsy for the last 10
years. He was admitted to the emergency department for leg pain, cramping in the
hands and legs, and agitation multiple times within the last six months. He was
prescribed antidepressant and antipsychotic medications. He had a blood pressure
of 150/90?mmHg, diffuse abdominal tenderness, and abdominal muscle rigidity on
physical examination. Pathological laboratory findings were as follows:
creatinine, 7.5?mg/dL, calcium, 3.7?mg/dL, alanine transaminase, 4349?U/L,
aspartate transaminase, 5237?U/L, creatine phosphokinase, 262.000?U/L, and
parathyroid hormone, 0?pg/mL. There were bilateral symmetrical calcifications in
basal ganglia and the cerebellum on computerized tomography. He was diagnosed as
primary hypoparathyroidism and acute kidney injury secondary to severe
rhabdomyolysis. Brain calcifications, although rare, should be considered in
dealing with patients with neurological symptoms, symmetrical cranial
calcifications, and calcium metabolism abnormalities.