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10.1186/s12916-016-0601-y http://scihub22266oqcxt.onion/10.1186/s12916-016-0601-y C4802632!4802632!27001187     free     free     free Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       BMC+Med 2016 ; 14 (ä): ä Nephropedia Template TP {{tp|p=27001187|t=2016. Zika virus and neurologic autoimmunity: the putative role of gangliosides |pdf=|usr=}}{{27001187}} gab.com Text Zika virus and neurologic autoimmunity: the putative role of gangliosides JO: BMC Med http://www.kidney.de/pget.php?&tw=1&pmid=27001187 #FOAvip An increasing number of severe neurological complications associated with Zika virus (ZIKV), chiefly Guillain-Barré syndrome (GBS) and primary microcephaly, have led the World Health Organization to declare a global health emergency. Molecular mimicry between glycolipids and surface molecules of infectious agents explain most of the cases of GBS preceded by infection, while a direct toxicity of ZIKV on neural cells has been raised as the main mechanism by which ZIKV induces microcephaly. Gangliosides are crucial in brain development, and their expression correlates with neurogenesis, synaptogenesis, synaptic transmission, and cell proliferation. Targeting the autoimmune response to gangliosides may represent an underexploited opportunity to examine the increased incidence of neurological complications related to ZIKV infection. Twit Text FOAVip Zika virus and neurologic autoimmunity: the putative role of gangliosides ????BMC Med http://www.kidney.de/pget.php?&tw=1&pmid=27001187 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27001187 #FOAvip English Wikipedia <ref>{{Cite pmid|27001187}}</ref> Zika virus and neurologic autoimmunity: the putative role of gangliosides #MMPMID27001187 Anaya JM; Ramirez-Santana C; Salgado-Castaneda I; Chang C; Ansari A; Gershwin ME BMC Med 2016[]; 14 (ä): ä PMID27001187show ga An increasing number of severe neurological complications associated with Zika virus (ZIKV), chiefly Guillain-Barré syndrome (GBS) and primary microcephaly, have led the World Health Organization to declare a global health emergency. Molecular mimicry between glycolipids and surface molecules of infectious agents explain most of the cases of GBS preceded by infection, while a direct toxicity of ZIKV on neural cells has been raised as the main mechanism by which ZIKV induces microcephaly. Gangliosides are crucial in brain development, and their expression correlates with neurogenesis, synaptogenesis, synaptic transmission, and cell proliferation. Targeting the autoimmune response to gangliosides may represent an underexploited opportunity to examine the increased incidence of neurological complications related to ZIKV infection. äZika virus and neurologic autoimmunity: the putative role of gangliosi(epmc).pdf DeepDyve Pubget Overpricing