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10.1016/j.immuni.2016.02.015 http://scihub22266oqcxt.onion/10.1016/j.immuni.2016.02.015 C4794754!4794754!26982353     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Immunity 2016 ; 44 (3): 450-62 Nephropedia Template TP {{tp|p=26982353|t=2016. Macrophages in tissue repair, regeneration, and fibrosis |pdf=|usr=}}{{26982353}} gab.com Text Macrophages in tissue repair, regeneration, and fibrosis JO: Immunity http://www.kidney.de/pget.php?&tw=1&pmid=26982353 #FOAvip Inflammatory monocytes and resident tissue macrophages are key regulators of tissue repair, regeneration, and fibrosis. Following tissue injury, monocytes and macrophages undergo marked phenotypic and functional changes to play critical roles during the initiation, maintenance, and resolution phases of tissue repair. Disturbances in macrophage function can lead to aberrant repair, with uncontrolled inflammatory mediator and growth factor production, deficient generation of anti-inflammatory macrophages, or failed communication between macrophages and epithelial cells, endothelial cells, fibroblasts, and stem or tissue progenitor cells all contributing to a state of persistent injury, which may lead to the development of pathological fibrosis. In this review, we discuss the mechanisms that instruct macrophages to adopt pro-inflammatory, pro-wound healing, pro-fibrotic, anti-inflammatory, anti-fibrotic, pro-resolving, and tissue regenerating phenotypes following injury, and highlight how some of these mechanisms and macrophage activation states could be exploited therapeutically. Twit Text FOAVip Macrophages in tissue repair, regeneration, and fibrosis ????Immunity http://www.kidney.de/pget.php?&tw=1&pmid=26982353 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26982353 #FOAvip English Wikipedia <ref>{{Cite pmid|26982353}}</ref> Macrophages in tissue repair, regeneration, and fibrosis #MMPMID26982353 Wynn TA; Vannella KM Immunity 2016[Mar]; 44 (3): 450-62 PMID26982353show ga Inflammatory monocytes and resident tissue macrophages are key regulators of tissue repair, regeneration, and fibrosis. Following tissue injury, monocytes and macrophages undergo marked phenotypic and functional changes to play critical roles during the initiation, maintenance, and resolution phases of tissue repair. Disturbances in macrophage function can lead to aberrant repair, with uncontrolled inflammatory mediator and growth factor production, deficient generation of anti-inflammatory macrophages, or failed communication between macrophages and epithelial cells, endothelial cells, fibroblasts, and stem or tissue progenitor cells all contributing to a state of persistent injury, which may lead to the development of pathological fibrosis. In this review, we discuss the mechanisms that instruct macrophages to adopt pro-inflammatory, pro-wound healing, pro-fibrotic, anti-inflammatory, anti-fibrotic, pro-resolving, and tissue regenerating phenotypes following injury, and highlight how some of these mechanisms and macrophage activation states could be exploited therapeutically. äMacrophages in tissue repair, regeneration, and fibrosis (epmc).pdf DeepDyve Pubget Overpricing