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10.1007/s10048-015-0469-6 http://scihub22266oqcxt.onion/10.1007/s10048-015-0469-6 C4794525!4794525!26660102     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Neurogenetics 2016 ; 17 (ä): 125-30 Nephropedia Template TP {{tp|p=26660102|t=2016. Slc20a2 is critical for maintaining a physiologic inorganic phosphate level in cerebrospinal fluid |pdf=|usr=}}{{26660102}} gab.com Text Slc20a2 is critical for maintaining a physiologic inorganic phosphate level in cerebrospinal fluid JO: Neurogenetics http://www.kidney.de/pget.php?&tw=1&pmid=26660102 #FOAvip Mutations in the SLC20A2-gene encoding the inorganic phosphate (Pi) transporter PiT2 can explain approximately 40 % of the familial cases of the rare neurodegenerative disorder primary familial brain calcification (Fahr?s disease). The disease characteristic, cerebrovascular-associated calcifications, is also present in Slc20a2-knockout (KO) mice. Little is known about the specific role(s) of PiT2 in the brain. Recent in vitro studies, however, suggest a role in regulation of the [Pi] in cerebrospinal fluid (CSF). We here show that Slc20a2-KO mice indeed have a high CSF [Pi] in agreement with a role of PiT2 in Pi export from the CSF. The implications in relation to disease mechanism are discussed. Twit Text FOAVip Slc20a2 is critical for maintaining a physiologic inorganic phosphate level in cerebrospinal fluid ????Neurogenetics http://www.kidney.de/pget.php?&tw=1&pmid=26660102 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26660102 #FOAvip English Wikipedia <ref>{{Cite pmid|26660102}}</ref> Slc20a2 is critical for maintaining a physiologic inorganic phosphate level in cerebrospinal fluid #MMPMID26660102 Jensen N; Autzen JK; Pedersen L Neurogenetics 2016[]; 17 (ä): 125-30 PMID26660102show ga Mutations in the SLC20A2-gene encoding the inorganic phosphate (Pi) transporter PiT2 can explain approximately 40 % of the familial cases of the rare neurodegenerative disorder primary familial brain calcification (Fahr?s disease). The disease characteristic, cerebrovascular-associated calcifications, is also present in Slc20a2-knockout (KO) mice. Little is known about the specific role(s) of PiT2 in the brain. Recent in vitro studies, however, suggest a role in regulation of the [Pi] in cerebrospinal fluid (CSF). We here show that Slc20a2-KO mice indeed have a high CSF [Pi] in agreement with a role of PiT2 in Pi export from the CSF. The implications in relation to disease mechanism are discussed. äSlc20a2 is critical for maintaining a physiologic inorganic phosphate (epmc).pdf DeepDyve Pubget Overpricing