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10.3389/fimmu.2016.00084 http://scihub22266oqcxt.onion/10.3389/fimmu.2016.00084 C4783396!4783396!27014264     free     free     free Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Front+Immunol 2016 ; 7 (ä): ä Nephropedia Template TP {{tp|p=27014264|t=2016. Bacterial Control of Pores Induced by the Type III Secretion System: Mind the Gap |pdf=|usr=}}{{27014264}} gab.com Text Bacterial Control of Pores Induced by the Type III Secretion System: Mind the Gap JO: Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=27014264 #FOAvip Type III secretion systems (T3SSs) are specialized secretion apparatus involved in the virulence of many Gram-negative pathogens, enabling the injection of bacterial type III effectors into host cells. The T3SS-dependent injection of effectors requires the insertion into host cell membranes of a pore-forming ?translocon,? whose effects on cell responses remain ill-defined. As opposed to pore-forming toxins that damage host cell plasma membranes and induce cell survival mechanisms, T3SS-dependent pore formation is transient, being regulated by cell membrane repair mechanisms or bacterial effectors. Here, we review host cell responses to pore formation induced by T3SSs associated with the loss of plasma membrane integrity and regulation of innate immunity. We will particularly focus on recent advances in mechanisms controlling pore formation and the activity of the T3SS linked to type III effectors or bacterial proteases. The implications of the regulation of the T3SS translocon activity during the infectious process will be discussed. Twit Text FOAVip Bacterial Control of Pores Induced by the Type III Secretion System: Mind the Gap ????Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=27014264 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27014264 #FOAvip English Wikipedia <ref>{{Cite pmid|27014264}}</ref> Bacterial Control of Pores Induced by the Type III Secretion System: Mind the Gap #MMPMID27014264 Guignot J; Tran Van Nhieu G Front Immunol 2016[]; 7 (ä): ä PMID27014264show ga Type III secretion systems (T3SSs) are specialized secretion apparatus involved in the virulence of many Gram-negative pathogens, enabling the injection of bacterial type III effectors into host cells. The T3SS-dependent injection of effectors requires the insertion into host cell membranes of a pore-forming ?translocon,? whose effects on cell responses remain ill-defined. As opposed to pore-forming toxins that damage host cell plasma membranes and induce cell survival mechanisms, T3SS-dependent pore formation is transient, being regulated by cell membrane repair mechanisms or bacterial effectors. Here, we review host cell responses to pore formation induced by T3SSs associated with the loss of plasma membrane integrity and regulation of innate immunity. We will particularly focus on recent advances in mechanisms controlling pore formation and the activity of the T3SS linked to type III effectors or bacterial proteases. The implications of the regulation of the T3SS translocon activity during the infectious process will be discussed. äBacterial Control of Pores Induced by the Type III Secretion System: M(epmc).pdf DeepDyve Pubget Overpricing