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10.1186/s12883-016-0557-8 http://scihub22266oqcxt.onion/10.1186/s12883-016-0557-8 C4782297!4782297!26951767     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       BMC+Neurol 2016 ; 16 (ä): ä Nephropedia Template TP {{tp|p=26951767|t=2016. Ischemic stroke in a young patient with Fahr?s disease: a case report |pdf=|usr=}}{{26951767}} gab.com Text Ischemic stroke in a young patient with Fahr s disease: a case report JO: BMC Neurol http://www.kidney.de/pget.php?&tw=1&pmid=26951767 #FOAvip Background: Fahr?s disease is a rare neurodegenerative disorder characterized by diffuse intracranial calcium deposition and associated cell loss mainly in bilateral basal ganglia and dentate nuclei of the cerebellum. Subarachnoid hemorrhage and epileptic syncope had been reported as acute presentation of Fahr?s disease. We here report a 36-year-old male Indonesian diagnosed as Fahr?s disease presenting with young-onset ischemic stroke. Case presentation: A 36-year-old male Indonesian without prior systemic disease or neurologic disorder presented with young-onset ischemic stroke involving the right posterior limb of internal capsule. Brain computed tomography and magnetic resonance imaging demonstrated symmetric calcifications in bilateral basal ganglia, internal capsules, cerebellar dentate nuclei, thalami, cerebral white matter, which were all consistent with Fahr?s disease. The laboratory studies excluded the presence of other pathologic processes leading to secondary intracranial calcification. Other young stroke surveys were unremarkable. After medical treatment and sustained physical rehabilitation for 3 months, he recovered to carry out daily activities independently. Conclusion: We present ischemic stroke in a young patient with sporadic Fahr?s disease. The differentiation between Fahr?s disease and Fahr?s syndrome is specially highlighted when brain CT exhibits diffuse, symmetric calcifications in bilateral basal ganglia, thalami, cerebellar dentate nuclei and cerebral white matter. The association between nonarteriosclerotic vascular calcification and cerebrovascular disease is worth special attention and further investigation. Twit Text FOAVip Ischemic stroke in a young patient with Fahr s disease: a case report ????BMC Neurol http://www.kidney.de/pget.php?&tw=1&pmid=26951767 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26951767 #FOAvip English Wikipedia <ref>{{Cite pmid|26951767}}</ref> Ischemic stroke in a young patient with Fahr?s disease: a case report #MMPMID26951767 Yang CS; Lo CP; Wu MC BMC Neurol 2016[]; 16 (ä): ä PMID26951767show ga Background: Fahr?s disease is a rare neurodegenerative disorder characterized by diffuse intracranial calcium deposition and associated cell loss mainly in bilateral basal ganglia and dentate nuclei of the cerebellum. Subarachnoid hemorrhage and epileptic syncope had been reported as acute presentation of Fahr?s disease. We here report a 36-year-old male Indonesian diagnosed as Fahr?s disease presenting with young-onset ischemic stroke. Case presentation: A 36-year-old male Indonesian without prior systemic disease or neurologic disorder presented with young-onset ischemic stroke involving the right posterior limb of internal capsule. Brain computed tomography and magnetic resonance imaging demonstrated symmetric calcifications in bilateral basal ganglia, internal capsules, cerebellar dentate nuclei, thalami, cerebral white matter, which were all consistent with Fahr?s disease. The laboratory studies excluded the presence of other pathologic processes leading to secondary intracranial calcification. Other young stroke surveys were unremarkable. After medical treatment and sustained physical rehabilitation for 3 months, he recovered to carry out daily activities independently. Conclusion: We present ischemic stroke in a young patient with sporadic Fahr?s disease. The differentiation between Fahr?s disease and Fahr?s syndrome is specially highlighted when brain CT exhibits diffuse, symmetric calcifications in bilateral basal ganglia, thalami, cerebellar dentate nuclei and cerebral white matter. The association between nonarteriosclerotic vascular calcification and cerebrovascular disease is worth special attention and further investigation. äIschemic stroke in a young patient with Fahr?s disease: a case report (epmc).pdf DeepDyve Pubget Overpricing