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10.1073/pnas.1519467113 http://scihub22266oqcxt.onion/10.1073/pnas.1519467113 C4780619!4780619!26884170     free     free     free Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Proc+Natl+Acad+Sci+U+S+A 2016 ; 113 (9): 2472-7 Nephropedia Template TP {{tp|p=26884170|t=2016. Gene control of tyrosine kinase TIE2 and vascular manifestations of infections |pdf=|usr=}}{{26884170}} gab.com Text Gene control of tyrosine kinase TIE2 and vascular manifestations of infections JO: Proc Natl Acad Sci U S A http://www.kidney.de/pget.php?&tw=1&pmid=26884170 #FOAvip Major infections, such as influenza and bacterial sepsis, kill millions of individuals yearly, most commonly from complications affecting the vasculature, such as acute respiratory distress syndrome. Poor outcomes from rare infections, such as Ebola virus disease, have also been linked to the vasculature. The basis for prominent vascular involvement in infectious syndromes remains poorly understood. The present work shows that humans exhibit common, yet highly consequential, genetic variation in the ability to sustain expression of a key homeostatic vascular receptor called tunica interna endothelial cell kinase 2. The results suggest that host determinants of the molecular vascular response to infection may have a heretofore underappreciated impact on clinical outcomes. They also suggest new means to identify at-risk individuals and personalize future therapies. Twit Text FOAVip Gene control of tyrosine kinase TIE2 and vascular manifestations of infections ????Proc Natl Acad Sci U S A http://www.kidney.de/pget.php?&tw=1&pmid=26884170 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26884170 #FOAvip English Wikipedia <ref>{{Cite pmid|26884170}}</ref> Gene control of tyrosine kinase TIE2 and vascular manifestations of infections #MMPMID26884170 Ghosh CC; David S; Zhang R; Berghelli A; Milam K; Higgins SJ; Hunter J; Mukherjee A; Wei Y; Tran M; Suber F; Kobzik L; Kain KC; Lu S; Santel A; Yano K; Guha P; Dumont DJ; Christiani DC; Parikh SM Proc Natl Acad Sci U S A 2016[Mar]; 113 (9): 2472-7 PMID26884170show ga Major infections, such as influenza and bacterial sepsis, kill millions of individuals yearly, most commonly from complications affecting the vasculature, such as acute respiratory distress syndrome. Poor outcomes from rare infections, such as Ebola virus disease, have also been linked to the vasculature. The basis for prominent vascular involvement in infectious syndromes remains poorly understood. The present work shows that humans exhibit common, yet highly consequential, genetic variation in the ability to sustain expression of a key homeostatic vascular receptor called tunica interna endothelial cell kinase 2. The results suggest that host determinants of the molecular vascular response to infection may have a heretofore underappreciated impact on clinical outcomes. They also suggest new means to identify at-risk individuals and personalize future therapies. äGene control of tyrosine kinase TIE2 and vascular manifestations of in(epmc).pdf DeepDyve Pubget Overpricing