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10.4049/jimmunol.1500801 http://scihub22266oqcxt.onion/10.4049/jimmunol.1500801 C4778549!4778549!26342103     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Immunol 2015 ; 195 (6): 2507-13 Nephropedia Template TP {{tp|p=26342103|t=2015. Control of regulatory T cell migration, function and homeostasis1 |pdf=|usr=}}{{26342103}} gab.com Text Control of regulatory T cell migration, function and homeostasis1 JO: J Immunol http://www.kidney.de/pget.php?&tw=1&pmid=26342103 #FOAvip Foxp3+ regulatory T (Treg) cells are essential for preventing autoimmunity and uncontrolled inflammation, and also modulate immune responses during infection and development of cancer. Accomplishing these tasks requires the widespread distribution of Treg cells in both lymphoid and non-lymphoid tissues, and the selective recruitment of Treg cells to different tissue sites has emerged as a key checkpoint that controlling tissue inflammation in autoimmunity, infection, cancer development and in the context of allograft acceptance or rejection. Additionally, Treg cells are functionally diverse, and it has become clear that some of this diversity segregates with Treg cell localization to particular tissue sites. In this article, I will review progress in understanding the mechanisms of Treg cell trafficking, and discuss factors controlling their homeostatic maintenance and function in distinct tissue sites. Twit Text FOAVip Control of regulatory T cell migration, function and homeostasis1 ????J Immunol http://www.kidney.de/pget.php?&tw=1&pmid=26342103 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26342103 #FOAvip English Wikipedia <ref>{{Cite pmid|26342103}}</ref> Control of regulatory T cell migration, function and homeostasis1 #MMPMID26342103 Campbell DJ J Immunol 2015[Sep]; 195 (6): 2507-13 PMID26342103show ga Foxp3+ regulatory T (Treg) cells are essential for preventing autoimmunity and uncontrolled inflammation, and also modulate immune responses during infection and development of cancer. Accomplishing these tasks requires the widespread distribution of Treg cells in both lymphoid and non-lymphoid tissues, and the selective recruitment of Treg cells to different tissue sites has emerged as a key checkpoint that controlling tissue inflammation in autoimmunity, infection, cancer development and in the context of allograft acceptance or rejection. Additionally, Treg cells are functionally diverse, and it has become clear that some of this diversity segregates with Treg cell localization to particular tissue sites. In this article, I will review progress in understanding the mechanisms of Treg cell trafficking, and discuss factors controlling their homeostatic maintenance and function in distinct tissue sites. äControl of regulatory T cell migration, function and homeostasis1 (epmc).pdf DeepDyve Pubget Overpricing