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10.1159/000441715 http://scihub22266oqcxt.onion/10.1159/000441715 C4777904!4777904!26955632     free     free     free Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Case+Rep+Nephrol+Dial 2015 ; 5 (3): 204-12 Nephropedia Template TP {{tp|p=26955632|t=2015. Macrophage Infiltration into the Glomeruli in Lipoprotein Glomerulopathy |pdf=|usr=}}{{26955632}} gab.com Text Macrophage Infiltration into the Glomeruli in Lipoprotein Glomerulopathy JO: Case Rep Nephrol Dial http://www.kidney.de/pget.php?&tw=1&pmid=26955632 #FOAvip Lipoprotein glomerulopathy (LPG) is characterized by histopathological features showing intra-glomerular lipoprotein thrombi and type III hyperlipoproteinemia (HLP), with heterozygote mutation of apolipoprotein (apo) E gene. On the other hand, as another renal lipidosis with type III HLP, apoE2 homozygote-related glomerulopathy (apoE2-GN) showing foamy macrophages has been reported. The case of a 25-year-old man who had LPG by clinical behavior and gene analysis, but demonstrated atypical histopathological features with a substantial amount of foamy macrophage infiltration in the glomeruli, is presented. The combination of alleles for apoE Tokyo/Maebashi and classical apoE2 (Arg158Cys) was inferred to be the leading cause of the unique renal pathology with lipoprotein thrombi and foamy macrophages. In addition, foamy macrophages infiltrated some part of the apoE-positive region within the glomerulus, but did not exist in lipoprotein thrombi despite apoE positivity, suggesting that properties of apoE are crucial in the development of LPG rather than macrophage function. This case provides important information related to the pathogenesis of LPG and apoE2-GN. Twit Text FOAVip Macrophage Infiltration into the Glomeruli in Lipoprotein Glomerulopathy ????Case Rep Nephrol Dial http://www.kidney.de/pget.php?&tw=1&pmid=26955632 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26955632 #FOAvip English Wikipedia <ref>{{Cite pmid|26955632}}</ref> Macrophage Infiltration into the Glomeruli in Lipoprotein Glomerulopathy #MMPMID26955632 Takasaki S; Maeda K; Joh K; Yamakage S; Fukase S; Takahashi T; Suzuki M; Matsunaga A; Saito T Case Rep Nephrol Dial 2015[Sep]; 5 (3): 204-12 PMID26955632show ga Lipoprotein glomerulopathy (LPG) is characterized by histopathological features showing intra-glomerular lipoprotein thrombi and type III hyperlipoproteinemia (HLP), with heterozygote mutation of apolipoprotein (apo) E gene. On the other hand, as another renal lipidosis with type III HLP, apoE2 homozygote-related glomerulopathy (apoE2-GN) showing foamy macrophages has been reported. The case of a 25-year-old man who had LPG by clinical behavior and gene analysis, but demonstrated atypical histopathological features with a substantial amount of foamy macrophage infiltration in the glomeruli, is presented. The combination of alleles for apoE Tokyo/Maebashi and classical apoE2 (Arg158Cys) was inferred to be the leading cause of the unique renal pathology with lipoprotein thrombi and foamy macrophages. In addition, foamy macrophages infiltrated some part of the apoE-positive region within the glomerulus, but did not exist in lipoprotein thrombi despite apoE positivity, suggesting that properties of apoE are crucial in the development of LPG rather than macrophage function. This case provides important information related to the pathogenesis of LPG and apoE2-GN. äMacrophage Infiltration into the Glomeruli in Lipoprotein Glomerulopat(epmc).pdf DeepDyve Pubget Overpricing