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10.1053/j.ajkd.2012.01.031 http://scihub22266oqcxt.onion/10.1053/j.ajkd.2012.01.031 C4776048!4776048!22901631     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Am+J+Kidney+Dis 2012 ; 60 (3): 492-7 Nephropedia Template TP {{tp|p=22901631|t=2012. A Physiologic-Based Approach to the Treatment of a Patient With Hypokalemia |pdf=|usr=}}{{22901631}} gab.com Text A Physiologic-Based Approach to the Treatment of a Patient With Hypokalemia JO: Am J Kidney Dis http://www.kidney.de/pget.php?&tw=1&pmid=22901631 #FOAvip Hypokalemia is common, and can be associated with serious adverse consequences including paralysis, ileus, cardiac arrhythmias, and death. As a result, the body maintains serum potassium concentration within very narrow limits via tightly regulated feedback and feed-forward systems. Whereas the consequences of symptomatic hypokalemia and severe potassium depletion are well appreciated, chronic mild hypokalemia can accelerate the progression of chronic kidney disease, exacerbate systemic hypertension, and increase mortality. Persistent hypokalemia may reflect total body potassium depletion or increased renal potassium clearance. In a patient with simple potassium depletion, potassium replacement therapy should correct serum potassium concentration, but may have little effect when renal potassium clearance is abnormally increased from potassium wasting. In such cases addition of potassium sparing diuretics might be helpful. Serum potassium concentration is an inaccurate marker of total body potassium deficit. Mild hypokalemia may be associated with significant total body potassium deficits and conversely, total body potassium stores can be normal in hypokalemia due to redistribution. The speed and extent of potassium replacement should be dictated by the clinical picture and guided by frequent reassessment of serum potassium concentration. The goals of therapy should be to correct any potassium deficit if present without provoking hyperkalemia. Oral replacement is preferred except when there is no functioning bowel or in the setting of EKG changes, neurological symptoms, cardiac ischemia, or digitalis therapy. Twit Text FOAVip A Physiologic-Based Approach to the Treatment of a Patient With Hypokalemia ????Am J Kidney Dis http://www.kidney.de/pget.php?&tw=1&pmid=22901631 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=22901631 #FOAvip English Wikipedia <ref>{{Cite pmid|22901631}}</ref> A Physiologic-Based Approach to the Treatment of a Patient With Hypokalemia #MMPMID22901631 Asmar A; Mohandas R; Wingo CS Am J Kidney Dis 2012[Sep]; 60 (3): 492-7 PMID22901631show ga Hypokalemia is common, and can be associated with serious adverse consequences including paralysis, ileus, cardiac arrhythmias, and death. As a result, the body maintains serum potassium concentration within very narrow limits via tightly regulated feedback and feed-forward systems. Whereas the consequences of symptomatic hypokalemia and severe potassium depletion are well appreciated, chronic mild hypokalemia can accelerate the progression of chronic kidney disease, exacerbate systemic hypertension, and increase mortality. Persistent hypokalemia may reflect total body potassium depletion or increased renal potassium clearance. In a patient with simple potassium depletion, potassium replacement therapy should correct serum potassium concentration, but may have little effect when renal potassium clearance is abnormally increased from potassium wasting. In such cases addition of potassium sparing diuretics might be helpful. Serum potassium concentration is an inaccurate marker of total body potassium deficit. Mild hypokalemia may be associated with significant total body potassium deficits and conversely, total body potassium stores can be normal in hypokalemia due to redistribution. The speed and extent of potassium replacement should be dictated by the clinical picture and guided by frequent reassessment of serum potassium concentration. The goals of therapy should be to correct any potassium deficit if present without provoking hyperkalemia. Oral replacement is preferred except when there is no functioning bowel or in the setting of EKG changes, neurological symptoms, cardiac ischemia, or digitalis therapy. äA Physiologic-Based Approach to the Treatment of a Patient With Hypoka(epmc).pdf DeepDyve Pubget Overpricing