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10.3892/br.2016.578 http://scihub22266oqcxt.onion/10.3892/br.2016.578 C4774315!4774315!26998262     free     free     free       Biomed+Rep 2016 ; 4 (3): 269-74 Nephropedia Template TP {{tp|p=26998262|t=2016. Epithelial-mesenchymal transition in liver fibrosis |pdf=|usr=}}{{26998262}} gab.com Text Epithelial-mesenchymal transition in liver fibrosis JO: Biomed Rep http://www.kidney.de/pget.php?&tw=1&pmid=26998262 #FOAvip Liver fibrosis is the result of a sustained wound healing response to sustained chronic liver injury, which includes viral, alcoholic and autoimmune hepatitis. Hepatic regeneration is the dominant outcome of liver damage. The outcomes of successful repair are the replacement of dead epithelial cells with healthy epithelial cells, and reconstruction of the normal hepatic structure and function. Prevention of the development of epithelial-mesenchymal transition (EMT) may control and even reverse liver fibrosis. EMT is a critical process for an epithelial cell to undergo a conversion to a mesenchymal phenotype, and is believed to be an inflammation-induced response, which may have a central role in liver fibrosis. The origin of fibrogenic cells in liver fibrosis remains controversial. Numerous studies have investigated the origin of all fibrogenic cells within the liver and the mechanism of the signaling pathways that lead to the activation of EMT programs during numerous chronic liver diseases. The present study aimed to summarize the evidence to explain the possible role of EMT in liver fibrosis. Twit Text FOAVip Epithelial-mesenchymal transition in liver fibrosis ????Biomed Rep http://www.kidney.de/pget.php?&tw=1&pmid=26998262 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26998262 #FOAvip English Wikipedia <ref>{{Cite pmid|26998262}}</ref> Epithelial-mesenchymal transition in liver fibrosis #MMPMID26998262 ZHAO YL; ZHU RT; SUN YL Biomed Rep 2016[Mar]; 4 (3): 269-74 PMID26998262show ga Liver fibrosis is the result of a sustained wound healing response to sustained chronic liver injury, which includes viral, alcoholic and autoimmune hepatitis. Hepatic regeneration is the dominant outcome of liver damage. The outcomes of successful repair are the replacement of dead epithelial cells with healthy epithelial cells, and reconstruction of the normal hepatic structure and function. Prevention of the development of epithelial-mesenchymal transition (EMT) may control and even reverse liver fibrosis. EMT is a critical process for an epithelial cell to undergo a conversion to a mesenchymal phenotype, and is believed to be an inflammation-induced response, which may have a central role in liver fibrosis. The origin of fibrogenic cells in liver fibrosis remains controversial. Numerous studies have investigated the origin of all fibrogenic cells within the liver and the mechanism of the signaling pathways that lead to the activation of EMT programs during numerous chronic liver diseases. The present study aimed to summarize the evidence to explain the possible role of EMT in liver fibrosis. äEpithelial-mesenchymal transition in liver fibrosis (epmc).pdf DeepDyve Pubget Overpricing