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10.3390/jcm5020026 http://scihub22266oqcxt.onion/10.3390/jcm5020026 C4773782!4773782!26901232     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Clin+Med 2016 ; 5 (2): ä Nephropedia Template TP {{tp|p=26901232|t=2016. Homeostatic Signaling by Cell?Cell Junctions and Its Dysregulation during Cancer Progression |pdf=|usr=}}{{26901232}} gab.com Text Homeostatic Signaling by Cell Cell Junctions and Its Dysregulation during Cancer Progression JO: J Clin Med http://www.kidney.de/pget.php?&tw=1&pmid=26901232 #FOAvip The transition of sessile epithelial cells to a migratory, mesenchymal phenotype is essential for metazoan development and tissue repair, but this program is exploited by tumor cells in order to escape the confines of the primary organ site, evade immunosurveillance, and resist chemo-radiation. In addition, epithelial-to-mesenchymal transition (EMT) confers stem-like properties that increase efficiency of colonization of distant organs. This review evaluates the role of cell?cell junctions in suppressing EMT and maintaining a quiescent epithelium. We discuss the conflicting data on junctional signaling in cancer and recent developments that resolve some of these conflicts. We focus on evidence from breast cancer, but include other organ sites where appropriate. Current and potential strategies for inhibition of EMT are discussed. Twit Text FOAVip Homeostatic Signaling by Cell Cell Junctions and Its Dysregulation during Cancer Progression ????J Clin Med http://www.kidney.de/pget.php?&tw=1&pmid=26901232 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26901232 #FOAvip English Wikipedia <ref>{{Cite pmid|26901232}}</ref> Homeostatic Signaling by Cell?Cell Junctions and Its Dysregulation during Cancer Progression #MMPMID26901232 Yu Y; Elble RC J Clin Med 2016[Feb]; 5 (2): ä PMID26901232show ga The transition of sessile epithelial cells to a migratory, mesenchymal phenotype is essential for metazoan development and tissue repair, but this program is exploited by tumor cells in order to escape the confines of the primary organ site, evade immunosurveillance, and resist chemo-radiation. In addition, epithelial-to-mesenchymal transition (EMT) confers stem-like properties that increase efficiency of colonization of distant organs. This review evaluates the role of cell?cell junctions in suppressing EMT and maintaining a quiescent epithelium. We discuss the conflicting data on junctional signaling in cancer and recent developments that resolve some of these conflicts. We focus on evidence from breast cancer, but include other organ sites where appropriate. Current and potential strategies for inhibition of EMT are discussed. äHomeostatic Signaling by Cell?Cell Junctions and Its Dysregulation dur(epmc).pdf DeepDyve Pubget Overpricing