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10.1155/2016/2732705 http://scihub22266oqcxt.onion/10.1155/2016/2732705 C4773551!4773551!26989421     free     free     free Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Stem+Cells+Int 2016 ; 2016 (ä): ä Nephropedia Template TP {{tp|p=26989421|t=2016. Noncoding RNAs in Tumor Epithelial-to-Mesenchymal Transition |pdf=|usr=}}{{26989421}} gab.com Text Noncoding RNAs in Tumor Epithelial-to-Mesenchymal Transition JO: Stem Cells Int http://www.kidney.de/pget.php?&tw=1&pmid=26989421 #FOAvip Epithelial-derived tumor cells acquire the capacity for epithelial-to-mesenchymal transition (EMT), which enables them to invade adjacent tissues and/or metastasize to distant organs. Cancer metastasis is the main cause of cancer-related death. Molecular mechanisms involved in the switch from an epithelial phenotype to mesenchymal status are complicated and are controlled by a variety of signaling pathways. Recently, a set of noncoding RNAs (ncRNAs), including miRNAs and long noncoding RNAs (lncRNAs), were found to modulate gene expressions at either transcriptional or posttranscriptional levels. These ncRNAs are involved in EMT through their interplay with EMT-related transcription factors (EMT-TFs) and EMT-associated signaling. Reciprocal regulatory interactions between lncRNAs and miRNAs further increase the complexity of the regulation of gene expression and protein translation. In this review, we discuss recent findings regarding EMT-regulating ncRNAs and their associated signaling pathways involved in cancer progression. Twit Text FOAVip Noncoding RNAs in Tumor Epithelial-to-Mesenchymal Transition ????Stem Cells Int http://www.kidney.de/pget.php?&tw=1&pmid=26989421 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26989421 #FOAvip English Wikipedia <ref>{{Cite pmid|26989421}}</ref> Noncoding RNAs in Tumor Epithelial-to-Mesenchymal Transition #MMPMID26989421 Lin CW; Lin PY; Yang PC Stem Cells Int 2016[]; 2016 (ä): ä PMID26989421show ga Epithelial-derived tumor cells acquire the capacity for epithelial-to-mesenchymal transition (EMT), which enables them to invade adjacent tissues and/or metastasize to distant organs. Cancer metastasis is the main cause of cancer-related death. Molecular mechanisms involved in the switch from an epithelial phenotype to mesenchymal status are complicated and are controlled by a variety of signaling pathways. Recently, a set of noncoding RNAs (ncRNAs), including miRNAs and long noncoding RNAs (lncRNAs), were found to modulate gene expressions at either transcriptional or posttranscriptional levels. These ncRNAs are involved in EMT through their interplay with EMT-related transcription factors (EMT-TFs) and EMT-associated signaling. Reciprocal regulatory interactions between lncRNAs and miRNAs further increase the complexity of the regulation of gene expression and protein translation. In this review, we discuss recent findings regarding EMT-regulating ncRNAs and their associated signaling pathways involved in cancer progression. äNoncoding RNAs in Tumor Epithelial-to-Mesenchymal Transition (epmc).pdf DeepDyve Pubget Overpricing