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10.4331/wjbc.v7.i1.44 http://scihub22266oqcxt.onion/10.4331/wjbc.v7.i1.44 C4768124!4768124!26981195     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       World+J+Biol+Chem 2016 ; 7 (1): 44-63 Nephropedia Template TP {{tp|p=26981195|t=2016. New insights into sodium transport regulation in the distal nephron: Role of G-protein coupled receptors |pdf=|usr=}}{{26981195}} gab.com Text New insights into sodium transport regulation in the distal nephron: Role of G-protein coupled receptors JO: World J Biol Chem http://www.kidney.de/pget.php?&tw=1&pmid=26981195 #FOAvip The renal handling of Na+ balance is a major determinant of the blood pressure (BP) level. The inability of the kidney to excrete the daily load of Na+ represents the primary cause of chronic hypertension. Among the different segments that constitute the nephron, those present in the distal part (i.e., the cortical thick ascending limb, the distal convoluted tubule, the connecting and collecting tubules) play a central role in the fine-tuning of renal Na+ excretion and are the target of many different regulatory processes that modulate Na+ retention more or less efficiently. G-protein coupled receptors (GPCRs) are crucially involved in this regulation and could represent efficient pharmacological targets to control BP levels. In this review, we describe both classical and novel GPCR-dependent regulatory systems that have been shown to modulate renal Na+ absorption in the distal nephron. In addition to the multiplicity of the GPCR that regulate Na+ excretion, this review also highlights the complexity of these different pathways, and the connections between them. Twit Text FOAVip New insights into sodium transport regulation in the distal nephron: Role of G-protein coupled receptors ????World J Biol Chem http://www.kidney.de/pget.php?&tw=1&pmid=26981195 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26981195 #FOAvip English Wikipedia <ref>{{Cite pmid|26981195}}</ref> New insights into sodium transport regulation in the distal nephron: Role of G-protein coupled receptors #MMPMID26981195 Morla L; Edwards A; Crambert G World J Biol Chem 2016[Feb]; 7 (1): 44-63 PMID26981195show ga The renal handling of Na+ balance is a major determinant of the blood pressure (BP) level. The inability of the kidney to excrete the daily load of Na+ represents the primary cause of chronic hypertension. Among the different segments that constitute the nephron, those present in the distal part (i.e., the cortical thick ascending limb, the distal convoluted tubule, the connecting and collecting tubules) play a central role in the fine-tuning of renal Na+ excretion and are the target of many different regulatory processes that modulate Na+ retention more or less efficiently. G-protein coupled receptors (GPCRs) are crucially involved in this regulation and could represent efficient pharmacological targets to control BP levels. In this review, we describe both classical and novel GPCR-dependent regulatory systems that have been shown to modulate renal Na+ absorption in the distal nephron. In addition to the multiplicity of the GPCR that regulate Na+ excretion, this review also highlights the complexity of these different pathways, and the connections between them. äNew insights into sodium transport regulation in the distal nephron: R(epmc).pdf DeepDyve Pubget Overpricing