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10.1038/srep22251 http://scihub22266oqcxt.onion/10.1038/srep22251 C4766502!4766502!26911834     free     free     free Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Sci+Rep 2016 ; 6 (ä): ä Nephropedia Template TP {{tp|p=26911834|t=2016. Resveratrol ameliorates fibrosis and inflammation in a mouse model of nonalcoholic steatohepatitis |pdf=|usr=}}{{26911834}} gab.com Text Resveratrol ameliorates fibrosis and inflammation in a mouse model of nonalcoholic steatohepatitis JO: Sci Rep http://www.kidney.de/pget.php?&tw=1&pmid=26911834 #FOAvip The natural polyphenol compound resveratrol (RSV) is considered to have a broad spectrum of beneficial biological activities upon human health. However, the exact effect of RSV on steatosis (a phenotype of non-alcoholic fatty liver [NAFL]) or fibrosis and inflammation (major phenotypes of non-alcoholic steatohepatitis [NASH]) is not known. Our data showed that administration of RSV (2 or 20?mg/kg/day) did not suppress steatosis in a high-fat diet-induced model of NAFL in mice. In contrast, identical concentrations of RSV dramatically inhibited inflammation and fibrosis in a low-dose lipopolysaccharide-induced model of NASH. These data suggested that RSV administration-mediated improvement of inflammation and fibrosis was due to the inhibition of LPS reactivity controlled by CD14 expression in Kupffer cells. These findings suggest that RSV could be a candidate agent for the treatment of NASH. Twit Text FOAVip Resveratrol ameliorates fibrosis and inflammation in a mouse model of nonalcoholic steatohepatitis ????Sci Rep http://www.kidney.de/pget.php?&tw=1&pmid=26911834 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26911834 #FOAvip English Wikipedia <ref>{{Cite pmid|26911834}}</ref> Resveratrol ameliorates fibrosis and inflammation in a mouse model of nonalcoholic steatohepatitis #MMPMID26911834 Kessoku T; Imajo K; Honda Y; Kato T; Ogawa Y; Tomeno W; Kato S; Mawatari H; Fujita K; Yoneda M; Nagashima Y; Saito S; Wada K; Nakajima A Sci Rep 2016[]; 6 (ä): ä PMID26911834show ga The natural polyphenol compound resveratrol (RSV) is considered to have a broad spectrum of beneficial biological activities upon human health. However, the exact effect of RSV on steatosis (a phenotype of non-alcoholic fatty liver [NAFL]) or fibrosis and inflammation (major phenotypes of non-alcoholic steatohepatitis [NASH]) is not known. Our data showed that administration of RSV (2 or 20?mg/kg/day) did not suppress steatosis in a high-fat diet-induced model of NAFL in mice. In contrast, identical concentrations of RSV dramatically inhibited inflammation and fibrosis in a low-dose lipopolysaccharide-induced model of NASH. These data suggested that RSV administration-mediated improvement of inflammation and fibrosis was due to the inhibition of LPS reactivity controlled by CD14 expression in Kupffer cells. These findings suggest that RSV could be a candidate agent for the treatment of NASH. äResveratrol ameliorates fibrosis and inflammation in a mouse model of (epmc).pdf DeepDyve Pubget Overpricing