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10.1007/s10585-015-9769-3 http://scihub22266oqcxt.onion/10.1007/s10585-015-9769-3 C4761368!4761368!26620208     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26620208&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Clin+Exp+Metastasis 2016 ; 33 (ä): 197-210 Nephropedia Template TP {{tp|p=26620208|t=2016. Anti-angiogenic alternatives to VEGF blockade |pdf=|usr=}}{{26620208}} gab.com Text Anti-angiogenic alternatives to VEGF blockade JO: Clin Exp Metastasis http://www.kidney.de/pget.php?&tw=1&pmid=26620208 #FOAvip Angiogenesis is a major requirement for tumour formation and development. Anti-angiogenic treatments aim to starve the tumour of nutrients and oxygen and also guard against metastasis. The main anti-angiogenic agents to date have focused on blocking the pro-angiogenic vascular endothelial growth factors (VEGFs). While this approach has seen some success and has provided a proof of principle that such anti-angiogenic agents can be used as treatment, the overall outcome of VEGF blockade has been somewhat disappointing. There is a current need for new strategies in inhibiting tumour angiogenesis; this article will review current and historical examples in blocking various membrane receptors and components of the extracellular matrix important in angiogenesis. Targeting these newly discovered pro-angiogenic proteins could provide novel strategies for cancer therapy. Twit Text FOAVip Anti-angiogenic alternatives to VEGF blockade ????Clin Exp Metastasis http://www.kidney.de/pget.php?&tw=1&pmid=26620208 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26620208 #FOAvip English Wikipedia <ref>{{Cite pmid|26620208}}</ref> Anti-angiogenic alternatives to VEGF blockade #MMPMID26620208 Khan KA; Bicknell R Clin Exp Metastasis 2016[]; 33 (ä): 197-210 PMID26620208show ga Angiogenesis is a major requirement for tumour formation and development. Anti-angiogenic treatments aim to starve the tumour of nutrients and oxygen and also guard against metastasis. The main anti-angiogenic agents to date have focused on blocking the pro-angiogenic vascular endothelial growth factors (VEGFs). While this approach has seen some success and has provided a proof of principle that such anti-angiogenic agents can be used as treatment, the overall outcome of VEGF blockade has been somewhat disappointing. There is a current need for new strategies in inhibiting tumour angiogenesis; this article will review current and historical examples in blocking various membrane receptors and components of the extracellular matrix important in angiogenesis. Targeting these newly discovered pro-angiogenic proteins could provide novel strategies for cancer therapy. äAnti-angiogenic alternatives to VEGF blockade (epmc).pdf DeepDyve Pubget Overpricing