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10.1590/S1677-5538.IBJU.2014.0090 http://scihub22266oqcxt.onion/10.1590/S1677-5538.IBJU.2014.0090 C4756947!4756947!26742979     free     free     free Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Int+Braz+J+Urol 2015 ; 41 (6): 1185-93 Nephropedia Template TP {{tp|p=26742979|t=2015. Protective effect of hydrogen sulfide on renal injury in the experimental unilateral ureteral obstruction |pdf=|usr=}}{{26742979}} gab.com Text Protective effect of hydrogen sulfide on renal injury in the experimental unilateral ureteral obstruction JO: Int Braz J Urol http://www.kidney.de/pget.php?&tw=1&pmid=26742979 #FOAvip Introduction/Objective:: Ureteral obstruction is a common pathology and causes kidney fibrosis and dysfunction at late period. In this present study, we investigated the antifibrotic and antiinflammatory effects of hydrogen sulfide on kidney damage after unilateral ureteral obstruction (UUO) in rats. Materials and Methods:: 24 rats were divided into four groups. Group 1 was control, group 2 was sham, group 3 included rats with UUO and group 4 rats with UUO which were given sodium hydrogen sulfide (NaHS)-exogenous donor of hydrogen sulfide (intraperitoneally 56?moL/kg/day). After 14 days, rats were killed and their kidneys were taken and blood analysis was performed. Tubular necrosis, mononuclear cell infiltration and interstitial fibrosis were determined histopathologically in a part of the kidneys; nitric oxide (NO), malondialdehyde (MDA) and reduced glutathione (GSH) levels were determined in the other part of the kidneys. Urea-creatinine levels were investigated by blood analysis. Statistical analyses were made by the Chi-square test and one-way analysis of variance (ANOVA). Results:: There was no significantly difference for urea-creatinine levels among groups. Pathologically, there was serious tubular necrosis and fibrosis in group 3 and there was significantly decreasing of tubular necrosis and fibrosis in group 4 (p<0.005). Also, there was significantly increase of NO and MDA levels and decrease of GSH levels in group 3 compared to other groups (p<0.005). Conclusions:: hydrogen sulfide prevents kidney damage with antioxidant and antiinflammatory effect. Twit Text FOAVip Protective effect of hydrogen sulfide on renal injury in the experimental unilateral ureteral obstruction ????Int Braz J Urol http://www.kidney.de/pget.php?&tw=1&pmid=26742979 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26742979 #FOAvip English Wikipedia <ref>{{Cite pmid|26742979}}</ref> Protective effect of hydrogen sulfide on renal injury in the experimental unilateral ureteral obstruction #MMPMID26742979 Dursun M; Otunctemur A; Ozbek E; Sahin S; Besiroglu H; Ozsoy OD; Cekmen M; Somay A; Ozbay N Int Braz J Urol 2015[Nov]; 41 (6): 1185-93 PMID26742979show ga Introduction/Objective:: Ureteral obstruction is a common pathology and causes kidney fibrosis and dysfunction at late period. In this present study, we investigated the antifibrotic and antiinflammatory effects of hydrogen sulfide on kidney damage after unilateral ureteral obstruction (UUO) in rats. Materials and Methods:: 24 rats were divided into four groups. Group 1 was control, group 2 was sham, group 3 included rats with UUO and group 4 rats with UUO which were given sodium hydrogen sulfide (NaHS)-exogenous donor of hydrogen sulfide (intraperitoneally 56?moL/kg/day). After 14 days, rats were killed and their kidneys were taken and blood analysis was performed. Tubular necrosis, mononuclear cell infiltration and interstitial fibrosis were determined histopathologically in a part of the kidneys; nitric oxide (NO), malondialdehyde (MDA) and reduced glutathione (GSH) levels were determined in the other part of the kidneys. Urea-creatinine levels were investigated by blood analysis. Statistical analyses were made by the Chi-square test and one-way analysis of variance (ANOVA). Results:: There was no significantly difference for urea-creatinine levels among groups. Pathologically, there was serious tubular necrosis and fibrosis in group 3 and there was significantly decreasing of tubular necrosis and fibrosis in group 4 (p<0.005). Also, there was significantly increase of NO and MDA levels and decrease of GSH levels in group 3 compared to other groups (p<0.005). Conclusions:: hydrogen sulfide prevents kidney damage with antioxidant and antiinflammatory effect. äProtective effect of hydrogen sulfide on renal injury in the experimen(epmc).pdf DeepDyve Pubget Overpricing