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10.1016/j.acthis.2015.12.011 http://scihub22266oqcxt.onion/10.1016/j.acthis.2015.12.011 C4755810!4755810 !26797190     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26797190 &cmd=llinks): Failed to open stream: HTTP request failed! 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Antioxidant diet and sex interact to regulate NOS isoform expression and glomerular mesangium proliferation in Zucker diabetic rat kidney |pdf=|usr=}}{{26797190 }} gab.com Text Antioxidant diet and sex interact to regulate NOS isoform expression and glomerular mesangium proliferation in Zucker diabetic rat kidney JO: Acta Histochem http://www.kidney.de/pget.php?&tw=1&pmid=26797190 #FOAvip Oxidative stress contributes substantially to the pathophysiology of diabetic nephropathy (DN). Consumption of an antioxidant-fortified (AO) diet from an early age prevents or delays later development of DN in the Zucker rat female with type 2 diabetes. We hypothesize this is due to effects on mesangial matrix and renal nitric oxide synthase (NOS) distribution and to sex-specific differences in NOS responses in the diabetic kidney. Total glomerular tuft area (GTA) and PAS-positive tuft area (PTA), endothelial (e), neuronal (n) and inducible (i) NOS were quantified in males and females on AO or regular (REG) diet at 6 and 20 weeks of age. eNOS was observed in glomeruli and tubules. nNOS predominantly localized to tubular epithelium in both cortex and medulla. iNOS was expressed in proximal and distal tubules and collecting ducts. Sex, diabetes duration and AO diet affected the distribution of the three isoforms. GTA and PTA increased with duration of hyperglycemia and showed a negative correlation with renal levels of all NOS isoforms. AO diet in both genders was associated with less PAS-positive staining and less mesangial expansion than the REG diet, an early increase in cortical iNOS in males, and sex-specific changes in cortical eNOS at 20 weeks. These effects of AO diet may contribute to sex-specific preservation of renal function in females. Twit Text FOAVip Antioxidant diet and sex interact to regulate NOS isoform expression and glomerular mesangium proliferation in Zucker diabetic rat kidney ????Acta Histochem http://www.kidney.de/pget.php?&tw=1&pmid=26797190 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26797190 #FOAvip English Wikipedia <ref>{{Cite pmid|26797190 }}</ref> Antioxidant diet and sex interact to regulate NOS isoform expression and glomerular mesangium proliferation in Zucker diabetic rat kidney #MMPMID26797190 Slyvka Y ; Malgor R ; Inman SR ; Ding J ; Heh V ; Nowak FV Acta Histochem 2016[Mar]; 118 (2 ): 183-93 PMID26797190 show ga Oxidative stress contributes substantially to the pathophysiology of diabetic nephropathy (DN). Consumption of an antioxidant-fortified (AO) diet from an early age prevents or delays later development of DN in the Zucker rat female with type 2 diabetes. We hypothesize this is due to effects on mesangial matrix and renal nitric oxide synthase (NOS) distribution and to sex-specific differences in NOS responses in the diabetic kidney. Total glomerular tuft area (GTA) and PAS-positive tuft area (PTA), endothelial (e), neuronal (n) and inducible (i) NOS were quantified in males and females on AO or regular (REG) diet at 6 and 20 weeks of age. eNOS was observed in glomeruli and tubules. nNOS predominantly localized to tubular epithelium in both cortex and medulla. iNOS was expressed in proximal and distal tubules and collecting ducts. Sex, diabetes duration and AO diet affected the distribution of the three isoforms. GTA and PTA increased with duration of hyperglycemia and showed a negative correlation with renal levels of all NOS isoforms. AO diet in both genders was associated with less PAS-positive staining and less mesangial expansion than the REG diet, an early increase in cortical iNOS in males, and sex-specific changes in cortical eNOS at 20 weeks. These effects of AO diet may contribute to sex-specific preservation of renal function in females. |Administration, Oral [MESH] |Animals [MESH] |Antioxidants/*administration & dosage [MESH] |Diabetes Mellitus, Type 2/complications/diet therapy/*enzymology [MESH] |Diabetic Nephropathies/diet therapy/*enzymology/etiology [MESH] |Diet [MESH] |Female [MESH] |Isoenzymes/metabolism [MESH] |Kidney Glomerulus/enzymology/pathology [MESH] |Male [MESH] |Mesangial Cells/*enzymology [MESH] |Nitric Oxide Synthase Type I/*metabolism [MESH] |Nitric Oxide Synthase Type II/*metabolism [MESH] |Nitric Oxide Synthase Type III/*metabolism [MESH] |Rats, Zucker [MESH]Antioxidant diet and sex interact to regulate NOS isoform expression a(epmc).pdf DeepDyve Pubget Overpricing