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10.1051/parasite/2016006 http://scihub22266oqcxt.onion/10.1051/parasite/2016006 C4754534!4754534!26880421     free     free     free Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Parasite 2016 ; 23 (ä): ä Nephropedia Template TP {{tp|p=26880421|t=2016. A review of the proposed role of neutrophils in rodent amebic liver abscess models |pdf=|usr=}}{{26880421}} gab.com Text A review of the proposed role of neutrophils in rodent amebic liver abscess models JO: Parasite http://www.kidney.de/pget.php?&tw=1&pmid=26880421 #FOAvip Host invasion by Entamoeba histolytica, the pathogenic agent of amebiasis, can lead to the development of amebic liver abscess (ALA). Due to the difficulty of exploring host and amebic factors involved in the pathogenesis of ALA in humans, most studies have been conducted with animal models (e.g., mice, gerbils, and hamsters). Histopathological findings reveal that the chronic phase of ALA in humans corresponds to lytic or liquefactive necrosis, whereas in rodent models there is granulomatous inflammation. However, the use of animal models has provided important information on molecules and mechanisms of the host/parasite interaction. Hence, the present review discusses the possible role of neutrophils in the effector immune response in ALA in rodents. Properly activated neutrophils are probably successful in eliminating amebas through oxidative and non-oxidative mechanisms, including neutrophil degranulation, the generation of free radicals (O2?, H2O2, HOCl) and peroxynitrite, the activation of NADPH-oxidase and myeloperoxidase (MPO) enzymes, and the formation of neutrophil extracellular traps (NETs). On the other hand, if amebas are not eliminated in the early stages of infection, they trigger a prolonged and exaggerated inflammatory response that apparently causes ALAs. Genetic differences in animals and humans are likely to be key to a successful host immune response. Twit Text FOAVip A review of the proposed role of neutrophils in rodent amebic liver abscess models ????Parasite http://www.kidney.de/pget.php?&tw=1&pmid=26880421 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26880421 #FOAvip English Wikipedia <ref>{{Cite pmid|26880421}}</ref> A review of the proposed role of neutrophils in rodent amebic liver abscess models #MMPMID26880421 Campos-Rodríguez R; Gutiérrez-Meza M; Jarillo-Luna RA; Drago-Serrano ME; Abarca-Rojano E; Ventura-Juárez J; Cárdenas-Jaramillo LM; Pacheco-Yepez J Parasite 2016[]; 23 (ä): ä PMID26880421show ga Host invasion by Entamoeba histolytica, the pathogenic agent of amebiasis, can lead to the development of amebic liver abscess (ALA). Due to the difficulty of exploring host and amebic factors involved in the pathogenesis of ALA in humans, most studies have been conducted with animal models (e.g., mice, gerbils, and hamsters). Histopathological findings reveal that the chronic phase of ALA in humans corresponds to lytic or liquefactive necrosis, whereas in rodent models there is granulomatous inflammation. However, the use of animal models has provided important information on molecules and mechanisms of the host/parasite interaction. Hence, the present review discusses the possible role of neutrophils in the effector immune response in ALA in rodents. Properly activated neutrophils are probably successful in eliminating amebas through oxidative and non-oxidative mechanisms, including neutrophil degranulation, the generation of free radicals (O2?, H2O2, HOCl) and peroxynitrite, the activation of NADPH-oxidase and myeloperoxidase (MPO) enzymes, and the formation of neutrophil extracellular traps (NETs). On the other hand, if amebas are not eliminated in the early stages of infection, they trigger a prolonged and exaggerated inflammatory response that apparently causes ALAs. Genetic differences in animals and humans are likely to be key to a successful host immune response. äA review of the proposed role of neutrophils in rodent amebic liver ab(epmc).pdf DeepDyve Pubget Overpricing