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10.1155/2016/7867852 http://scihub22266oqcxt.onion/10.1155/2016/7867852 C4753052!4753052!26942201     free     free     free Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Biomed+Res+Int 2016 ; 2016 (ä): ä Nephropedia Template TP {{tp|p=26942201|t=2016. Advanced Glycation End Products Induce Obesity and Hepatosteatosis in CD-1 Wild-Type Mice |pdf=|usr=}}{{26942201}} gab.com Text Advanced Glycation End Products Induce Obesity and Hepatosteatosis in CD-1 Wild-Type Mice JO: Biomed Res Int http://www.kidney.de/pget.php?&tw=1&pmid=26942201 #FOAvip AGEs are a heterogeneous group of molecules formed from the nonenzymatic reaction of reducing sugars with free amino groups of proteins, lipids, and/or nucleic acids. AGEs have been shown to play a role in various conditions including cardiovascular disease and diabetes. In this study, we hypothesized that AGEs play a role in the ?multiple hit hypothesis? of nonalcoholic fatty liver disease (NAFLD) and contribute to the pathogenesis of hepatosteatosis. We measured the effects of various mouse chows containing high or low AGE in the presence of high or low fat content on mouse weight and epididymal fat pads. We also measured the effects of these chows on the inflammatory response by measuring cytokine levels and myeloperoxidase activity levels on liver supernatants. We observed significant differences in weight gain and epididymal fat pad weights in the high AGE-high fat (HAGE-HF) versus the other groups. Leptin, TNF-?, IL-6, and myeloperoxidase (MPO) levels were significantly higher in the HAGE-HF group. We conclude that a diet containing high AGEs in the presence of high fat induces weight gain and hepatosteatosis in CD-1 mice. This may represent a model to study the role of AGEs in the pathogenesis of hepatosteatosis and steatohepatitis. Twit Text FOAVip Advanced Glycation End Products Induce Obesity and Hepatosteatosis in CD-1 Wild-Type Mice ????Biomed Res Int http://www.kidney.de/pget.php?&tw=1&pmid=26942201 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26942201 #FOAvip English Wikipedia <ref>{{Cite pmid|26942201}}</ref> Advanced Glycation End Products Induce Obesity and Hepatosteatosis in CD-1 Wild-Type Mice #MMPMID26942201 Sayej WN; Knight III PR; Guo WA; Mullan B; Ohtake PJ; Davidson BA; Khan A; Baker RD; Baker SS Biomed Res Int 2016[]; 2016 (ä): ä PMID26942201show ga AGEs are a heterogeneous group of molecules formed from the nonenzymatic reaction of reducing sugars with free amino groups of proteins, lipids, and/or nucleic acids. AGEs have been shown to play a role in various conditions including cardiovascular disease and diabetes. In this study, we hypothesized that AGEs play a role in the ?multiple hit hypothesis? of nonalcoholic fatty liver disease (NAFLD) and contribute to the pathogenesis of hepatosteatosis. We measured the effects of various mouse chows containing high or low AGE in the presence of high or low fat content on mouse weight and epididymal fat pads. We also measured the effects of these chows on the inflammatory response by measuring cytokine levels and myeloperoxidase activity levels on liver supernatants. We observed significant differences in weight gain and epididymal fat pad weights in the high AGE-high fat (HAGE-HF) versus the other groups. Leptin, TNF-?, IL-6, and myeloperoxidase (MPO) levels were significantly higher in the HAGE-HF group. We conclude that a diet containing high AGEs in the presence of high fat induces weight gain and hepatosteatosis in CD-1 mice. This may represent a model to study the role of AGEs in the pathogenesis of hepatosteatosis and steatohepatitis. äAdvanced Glycation End Products Induce Obesity and Hepatosteatosis in (epmc).pdf DeepDyve Pubget Overpricing