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10.1155/2016/3543678 http://scihub22266oqcxt.onion/10.1155/2016/3543678 C4752998!4752998!26941801     free     free     free Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Stem+Cells+Int 2016 ; 2016 (ä): ä Nephropedia Template TP {{tp|p=26941801|t=2016. Molecular Mechanisms Underlying Peritoneal EMT and Fibrosis |pdf=|usr=}}{{26941801}} gab.com Text Molecular Mechanisms Underlying Peritoneal EMT and Fibrosis JO: Stem Cells Int http://www.kidney.de/pget.php?&tw=1&pmid=26941801 #FOAvip Peritoneal dialysis is a form of renal replacement alternative to the hemodialysis. During this treatment, the peritoneal membrane acts as a permeable barrier for exchange of solutes and water. Continual exposure to dialysis solutions, as well as episodes of peritonitis and hemoperitoneum, can cause acute/chronic inflammation and injury to the peritoneal membrane, which undergoes progressive fibrosis, angiogenesis, and vasculopathy, eventually leading to discontinuation of the peritoneal dialysis. Among the different events controlling this pathological process, epithelial to mesenchymal transition of mesothelial cells plays a main role in the induction of fibrosis and in subsequent functional deterioration of the peritoneal membrane. Here, the main extracellular inducers and cellular players are described. Moreover, signaling pathways acting during this process are elucidated, with emphasis on signals delivered by TGF-? family members and by Toll-like/IL-1? receptors. The understanding of molecular mechanisms underlying fibrosis of the peritoneal membrane has both a basic and a translational relevance, since it may be useful for setup of therapies aimed at counteracting the deterioration as well as restoring the homeostasis of the peritoneal membrane. Twit Text FOAVip Molecular Mechanisms Underlying Peritoneal EMT and Fibrosis ????Stem Cells Int http://www.kidney.de/pget.php?&tw=1&pmid=26941801 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26941801 #FOAvip English Wikipedia <ref>{{Cite pmid|26941801}}</ref> Molecular Mechanisms Underlying Peritoneal EMT and Fibrosis #MMPMID26941801 Strippoli R; Moreno-Vicente R; Battistelli C; Cicchini C; Noce V; Amicone L; Marchetti A; del Pozo MA; Tripodi M Stem Cells Int 2016[]; 2016 (ä): ä PMID26941801show ga Peritoneal dialysis is a form of renal replacement alternative to the hemodialysis. During this treatment, the peritoneal membrane acts as a permeable barrier for exchange of solutes and water. Continual exposure to dialysis solutions, as well as episodes of peritonitis and hemoperitoneum, can cause acute/chronic inflammation and injury to the peritoneal membrane, which undergoes progressive fibrosis, angiogenesis, and vasculopathy, eventually leading to discontinuation of the peritoneal dialysis. Among the different events controlling this pathological process, epithelial to mesenchymal transition of mesothelial cells plays a main role in the induction of fibrosis and in subsequent functional deterioration of the peritoneal membrane. Here, the main extracellular inducers and cellular players are described. Moreover, signaling pathways acting during this process are elucidated, with emphasis on signals delivered by TGF-? family members and by Toll-like/IL-1? receptors. The understanding of molecular mechanisms underlying fibrosis of the peritoneal membrane has both a basic and a translational relevance, since it may be useful for setup of therapies aimed at counteracting the deterioration as well as restoring the homeostasis of the peritoneal membrane. äMolecular Mechanisms Underlying Peritoneal EMT and Fibrosis (epmc).pdf DeepDyve Pubget Overpricing