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10.1038/ni.3332 http://scihub22266oqcxt.onion/10.1038/ni.3332 C4720992!4720992!26595889     free     free     free Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Nat+Immunol 2016 ; 17 (2): 179-86 Nephropedia Template TP {{tp|p=26595889|t=2016. Complementarity and redundancy of IL-22-producing innate lymphoid cells |pdf=|usr=}}{{26595889}} gab.com Text Complementarity and redundancy of IL-22-producing innate lymphoid cells JO: Nat Immunol http://www.kidney.de/pget.php?&tw=1&pmid=26595889 #FOAvip Intestinal T cells and group 3 innate lymphoid cells (ILC3) control the composition of the microbiota and gut immune responses. Within the gut there coexists ILC3 subsets which either express or lack the Natural cytoxicity receptor (NCR) NKp46. We identify here the transcriptional signature associated with the T-bet-dependent differentiation of NCR? ILC3 into NCR+ ILC3. Contrary to the prevailing view, we show by conditional deletion of the key ILC3 genes Stat3, Il22, Tbx21 and Mcl1 that NCR+ ILC3 were redundant for the control of mouse colonic infections with Citrobacter rodentium in the presence of T cells. However, NCR+ ILC3 were essential for cecum homeostasis. Our data show that interplay between intestinal ILC3 and adaptive lymphocytes results in robust complementary fail-safe mechanisms ensuring gut homeostasis. Twit Text FOAVip Complementarity and redundancy of IL-22-producing innate lymphoid cells ????Nat Immunol http://www.kidney.de/pget.php?&tw=1&pmid=26595889 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26595889 #FOAvip English Wikipedia <ref>{{Cite pmid|26595889}}</ref> Complementarity and redundancy of IL-22-producing innate lymphoid cells #MMPMID26595889 Rankin LC; Girard-Madoux MJH; Seillet C; Mielke LA; Kerdiles Y; Fenis A; Wieduwild E; Putoczki T; Mondot S; Lantz O; Demon D; Papenfuss AT; Smyth GK; Lamkanfi M; Carotta S; Renauld JC; Shi W; Carpentier S; Soos T; Arendt C; Ugolini S; Huntington ND; Belz GT; Vivier E Nat Immunol 2016[Feb]; 17 (2): 179-86 PMID26595889show ga Intestinal T cells and group 3 innate lymphoid cells (ILC3) control the composition of the microbiota and gut immune responses. Within the gut there coexists ILC3 subsets which either express or lack the Natural cytoxicity receptor (NCR) NKp46. We identify here the transcriptional signature associated with the T-bet-dependent differentiation of NCR? ILC3 into NCR+ ILC3. Contrary to the prevailing view, we show by conditional deletion of the key ILC3 genes Stat3, Il22, Tbx21 and Mcl1 that NCR+ ILC3 were redundant for the control of mouse colonic infections with Citrobacter rodentium in the presence of T cells. However, NCR+ ILC3 were essential for cecum homeostasis. Our data show that interplay between intestinal ILC3 and adaptive lymphocytes results in robust complementary fail-safe mechanisms ensuring gut homeostasis. äComplementarity and redundancy of IL-22-producing innate lymphoid cell(epmc).pdf DeepDyve Pubget Overpricing