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10.1016/j.coph.2015.10.009 http://scihub22266oqcxt.onion/10.1016/j.coph.2015.10.009 C4716877!4716877!26595306     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Curr+Opin+Pharmacol 2016 ; 26 (ä): 96-101 Nephropedia Template TP {{tp|p=26595306|t=2016. Neuro-immune Dysfunction During Brain Aging: New Insights in Microglial Cell Regulation |pdf=|usr=}}{{26595306}} gab.com Text Neuro-immune Dysfunction During Brain Aging: New Insights in Microglial Cell Regulation JO: Curr Opin Pharmacol http://www.kidney.de/pget.php?&tw=1&pmid=26595306 #FOAvip Microglia, the resident immune cells of the brain, are at the center of communication between the central nervous system and immune system. While these brain-immune interactions are balanced in healthy adulthood, the ability to maintain homeostasis during aging is impaired. Microglia develop a loss of integrated regulatory networks including aberrant signaling from other brain cells, immune sensors, and epigenetic modifiers. The low-grade chronic neuroinflammation associated with this dysfunctional activity likely contributes to cognitive deficits and susceptibility to age-related pathologies. A better understanding of the underlying mechanisms responsible for neuro-immune dysregulation with age is crucial for providing targeted therapeutic strategies to support brain repair and healthy aging. Twit Text FOAVip Neuro-immune Dysfunction During Brain Aging: New Insights in Microglial Cell Regulation ????Curr Opin Pharmacol http://www.kidney.de/pget.php?&tw=1&pmid=26595306 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26595306 #FOAvip English Wikipedia <ref>{{Cite pmid|26595306}}</ref> Neuro-immune Dysfunction During Brain Aging: New Insights in Microglial Cell Regulation #MMPMID26595306 Matt SM; Johnson RW Curr Opin Pharmacol 2016[Feb]; 26 (ä): 96-101 PMID26595306show ga Microglia, the resident immune cells of the brain, are at the center of communication between the central nervous system and immune system. While these brain-immune interactions are balanced in healthy adulthood, the ability to maintain homeostasis during aging is impaired. Microglia develop a loss of integrated regulatory networks including aberrant signaling from other brain cells, immune sensors, and epigenetic modifiers. The low-grade chronic neuroinflammation associated with this dysfunctional activity likely contributes to cognitive deficits and susceptibility to age-related pathologies. A better understanding of the underlying mechanisms responsible for neuro-immune dysregulation with age is crucial for providing targeted therapeutic strategies to support brain repair and healthy aging. äNeuro-immune Dysfunction During Brain Aging: New Insights in Microglia(epmc).pdf DeepDyve Pubget Overpricing