Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.1073/pnas.1515076112 http://scihub22266oqcxt.onion/10.1073/pnas.1515076112 C4702983!4702983!26655736     free     free     free Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Proc+Natl+Acad+Sci+U+S+A 2015 ; 112 (52): 15928-33 Nephropedia Template TP {{tp|p=26655736|t=2015. Interleukin-11 alters placentation and causes preeclampsia features in mice |pdf=|usr=}}{{26655736}} gab.com Text Interleukin-11 alters placentation and causes preeclampsia features in mice JO: Proc Natl Acad Sci U S A http://www.kidney.de/pget.php?&tw=1&pmid=26655736 #FOAvip Preeclampsia is an insidious disease, unique to humans, affecting ?8% of pregnancies. There are no early detection tests or pharmacological treatments. Impaired placentation is widely accepted to contribute to the pathogenesis. However, the mechanisms remain elusive, given the complications of studying first-trimester placental development in women. A major limitation for the study of new treatments is the lack of available animal models that recapitulate the full spectrum of preeclampsia features. We have developed a mouse model characterized by elevated levels of the cytokine Interleukin-11 (IL11). This study provides evidence of a novel pathway causative of preeclampsia features in vivo. It also provides a novel in vivo mouse model that is useful for preclinical studies to test potential therapeutics. Twit Text FOAVip Interleukin-11 alters placentation and causes preeclampsia features in mice ????Proc Natl Acad Sci U S A http://www.kidney.de/pget.php?&tw=1&pmid=26655736 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26655736 #FOAvip English Wikipedia <ref>{{Cite pmid|26655736}}</ref> Interleukin-11 alters placentation and causes preeclampsia features in mice #MMPMID26655736 Winship AL; Koga K; Menkhorst E; Van Sinderen M; Rainczuk K; Nagai M; Cuman C; Yap J; Zhang JG; Simmons D; Young MJ; Dimitriadis E Proc Natl Acad Sci U S A 2015[Dec]; 112 (52): 15928-33 PMID26655736show ga Preeclampsia is an insidious disease, unique to humans, affecting ?8% of pregnancies. There are no early detection tests or pharmacological treatments. Impaired placentation is widely accepted to contribute to the pathogenesis. However, the mechanisms remain elusive, given the complications of studying first-trimester placental development in women. A major limitation for the study of new treatments is the lack of available animal models that recapitulate the full spectrum of preeclampsia features. We have developed a mouse model characterized by elevated levels of the cytokine Interleukin-11 (IL11). This study provides evidence of a novel pathway causative of preeclampsia features in vivo. It also provides a novel in vivo mouse model that is useful for preclinical studies to test potential therapeutics. äInterleukin-11 alters placentation and causes preeclampsia features in(epmc).pdf DeepDyve Pubget Overpricing