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10.1182/asheducation-2015.1.160 http://scihub22266oqcxt.onion/10.1182/asheducation-2015.1.160 C4697437!4697437!26637716     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Hematology+Am+Soc+Hematol+Educ+Program 2015 ; 2015 (1): 160-7 Nephropedia Template TP {{tp|p=26637716|t=2015. Sickle cell trait diagnosis: clinical and social implications |pdf=|usr=}}{{26637716}} gab.com Text Sickle cell trait diagnosis: clinical and social implications JO: Hematology Am Soc Hematol Educ Program http://www.kidney.de/pget.php?&tw=1&pmid=26637716 #FOAvip The sickle hemoglobin (HbS) point mutation has independently undergone evolutionary selection at least five times in the world because of its overwhelming malarial protective effects in the heterozygous state. In 1949, homozygous Hb S or sickle cell disease (SCD) became the first inherited condition identified at the molecular level; however, since then, both SCD and heterozygous Hb S, sickle cell trait (SCT), have endured a long and complicated history. Hasty adoption of early mass screening programs for SCD, recent implementation of targeted screening mandates for SCT in athletics, and concerns about stigmatization have evoked considerable controversy regarding research and policy decisions for SCT. Although SCT is a largely protective condition in the context of malaria, clinical sequelae, such as exercise-related injury, renal complications, and venous thromboembolism can occur in affected carriers. The historical background of SCD and SCT has provided lessons about how research should be conducted in the modern era to minimize stigmatization, optimize study conclusions, and inform genetic counseling and policy decisions for SCT. Twit Text FOAVip Sickle cell trait diagnosis: clinical and social implications ????Hematology Am Soc Hematol Educ Program http://www.kidney.de/pget.php?&tw=1&pmid=26637716 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26637716 #FOAvip English Wikipedia <ref>{{Cite pmid|26637716}}</ref> Sickle cell trait diagnosis: clinical and social implications #MMPMID26637716 Naik RP; Haywood C Hematology Am Soc Hematol Educ Program 2015[Dec]; 2015 (1): 160-7 PMID26637716show ga The sickle hemoglobin (HbS) point mutation has independently undergone evolutionary selection at least five times in the world because of its overwhelming malarial protective effects in the heterozygous state. In 1949, homozygous Hb S or sickle cell disease (SCD) became the first inherited condition identified at the molecular level; however, since then, both SCD and heterozygous Hb S, sickle cell trait (SCT), have endured a long and complicated history. Hasty adoption of early mass screening programs for SCD, recent implementation of targeted screening mandates for SCT in athletics, and concerns about stigmatization have evoked considerable controversy regarding research and policy decisions for SCT. Although SCT is a largely protective condition in the context of malaria, clinical sequelae, such as exercise-related injury, renal complications, and venous thromboembolism can occur in affected carriers. The historical background of SCD and SCT has provided lessons about how research should be conducted in the modern era to minimize stigmatization, optimize study conclusions, and inform genetic counseling and policy decisions for SCT. äSickle cell trait diagnosis: clinical and social implications (epmc).pdf DeepDyve Pubget Overpricing