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Sevoflurane pretreatment enhance HIF-2? expression in mice after renal ischemia/reperfusion injury |pdf=|usr=}}{{26722509}} gab.com Text Sevoflurane pretreatment enhance HIF-2 expression in mice after renal ischemia/reperfusion injury JO: Int J Clin Exp Pathol http://www.kidney.de/pget.php?&tw=1&pmid=26722509 #FOAvip Ischemia/reperfusion (I/R) injury often occurs, which is one of the major causes of acute kidney injury, thus increasing in-hospital mortality. HIF-2? has a protective role against ischemia of the kidney. Renal ischemia/reperfusion under sevoflurane anesthesia resulted in drastic improvements in renal function. We hypothesized that underlying mechanism responsible for renal protection from sevoflurane pretreatment involves the upregulation of HIF-2?. Sevoflurane pretreatment were performed on WT and HIF-2? knockout mice before renal ischemia/reperfusion. Levels of blood urea nitrogen (BUN) and serum creatinine (Cr) were determined with a standard clinical automatic analyzer. The left kidneys were taken for morphological examination. Expression of HIF-2? in kidney tissue was examined by western blotting. In WT mice, group I/R injury had significantly higher BUN and Cr levels than group control, whereas group I/R + Sev had significantly lower BUN and Cr levels than group I/R injury. Renal HIF-2? expression levels were significantly higher in WT mice of group I/R + Sev than group control and group I/R. In HIF-2?-/- mice, group I/R + Sev showed much higher BUN and Cr levels and severer histological damage than group I/R and group control. Renal HIF-2? expression levels were significantly higher in WT mice of group I/R + Sev than group control and group I/R. Our findings suggested that HIF-2? might contribute to the beneficial effect of sevoflurane in renal ischemia/reperfusion injury. Twit Text FOAVip Sevoflurane pretreatment enhance HIF-2 expression in mice after renal ischemia/reperfusion injury ????Int J Clin Exp Pathol http://www.kidney.de/pget.php?&tw=1&pmid=26722509 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26722509 #FOAvip English Wikipedia <ref>{{Cite pmid|26722509}}</ref> Sevoflurane pretreatment enhance HIF-2? expression in mice after renal ischemia/reperfusion injury #MMPMID26722509 Zheng B; Zhan Q; Chen J; Xu H; He Z Int J Clin Exp Pathol 2015[]; 8 (10): 13114-9 PMID26722509show ga Ischemia/reperfusion (I/R) injury often occurs, which is one of the major causes of acute kidney injury, thus increasing in-hospital mortality. HIF-2? has a protective role against ischemia of the kidney. Renal ischemia/reperfusion under sevoflurane anesthesia resulted in drastic improvements in renal function. We hypothesized that underlying mechanism responsible for renal protection from sevoflurane pretreatment involves the upregulation of HIF-2?. Sevoflurane pretreatment were performed on WT and HIF-2? knockout mice before renal ischemia/reperfusion. Levels of blood urea nitrogen (BUN) and serum creatinine (Cr) were determined with a standard clinical automatic analyzer. The left kidneys were taken for morphological examination. Expression of HIF-2? in kidney tissue was examined by western blotting. In WT mice, group I/R injury had significantly higher BUN and Cr levels than group control, whereas group I/R + Sev had significantly lower BUN and Cr levels than group I/R injury. Renal HIF-2? expression levels were significantly higher in WT mice of group I/R + Sev than group control and group I/R. In HIF-2?-/- mice, group I/R + Sev showed much higher BUN and Cr levels and severer histological damage than group I/R and group control. Renal HIF-2? expression levels were significantly higher in WT mice of group I/R + Sev than group control and group I/R. Our findings suggested that HIF-2? might contribute to the beneficial effect of sevoflurane in renal ischemia/reperfusion injury. äSevoflurane pretreatment enhance HIF-2? expression in mice after renal(epmc).pdf DeepDyve Pubget Overpricing