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http://scihub22266oqcxt.onion/ C4680355!4680355 !26722410     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26722410 &cmd=llinks): Failed to open stream: HTTP request failed! 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However, little is known about the potential role of miR-98 in OSCC. Here, we found that miR-98 was downregulated in OSCC tissues and cell lines. Overexpression of miR-98 inhibited proliferation, colony formation, migration, and invasion of OSCC cells. IGF1R was identified as the potential target of miR-98 using dual luciferase assay, qRT-PCR and western blot. Furthermore, restoration of IGF1R remarkably reversed the tumor-suppressive effects of miR-98 on OSCC cells. Moreover, miR-98 expression was inversely correlated with IGF1R expression in 19 cases of OSCC. These findings suggest that miR-98 inhibits cancer cell growth and metastasis by direct targeting IGF1R, implicating miR-98 as a novel potential therapeutic target for OSCC. Twit Text FOAVip miR-98 suppresses tumor cell growth and metastasis by targeting IGF1R in oral squamous cell carcinoma ????Int J Clin Exp Pathol http://www.kidney.de/pget.php?&tw=1&pmid=26722410 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26722410 #FOAvip English Wikipedia <ref>{{Cite pmid|26722410 }}</ref> miR-98 suppresses tumor cell growth and metastasis by targeting IGF1R in oral squamous cell carcinoma #MMPMID26722410 Du Y ; Li Y ; Lv H ; Zhou S ; Sun Z ; Wang M Int J Clin Exp Pathol 2015[]; 8 (10 ): 12252-9 PMID26722410 show ga Increasing evidences indicate that dysregulation of miRNAs contributes to the pathogenesis of oral squamous cell carcinoma (OSCC). However, little is known about the potential role of miR-98 in OSCC. Here, we found that miR-98 was downregulated in OSCC tissues and cell lines. Overexpression of miR-98 inhibited proliferation, colony formation, migration, and invasion of OSCC cells. IGF1R was identified as the potential target of miR-98 using dual luciferase assay, qRT-PCR and western blot. Furthermore, restoration of IGF1R remarkably reversed the tumor-suppressive effects of miR-98 on OSCC cells. Moreover, miR-98 expression was inversely correlated with IGF1R expression in 19 cases of OSCC. These findings suggest that miR-98 inhibits cancer cell growth and metastasis by direct targeting IGF1R, implicating miR-98 as a novel potential therapeutic target for OSCC. |*Gene Expression Regulation, Neoplastic [MESH] |Carcinogenesis [MESH] |Carcinoma, Squamous Cell/genetics/*pathology [MESH] |Cell Line, Tumor [MESH] |Cell Movement [MESH] |Cell Proliferation [MESH] |Down-Regulation [MESH] |Humans [MESH] |MicroRNAs/*genetics [MESH] |Mouth Neoplasms/genetics/*pathology [MESH] |Neoplasm Invasiveness [MESH] |Neoplasm Metastasis [MESH] |Receptor, IGF Type 1 [MESH]miR-98 suppresses tumor cell growth and metastasis by targeting IGF1R (epmc).pdf DeepDyve Pubget Overpricing