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10.1016/j.heares.2015.06.005 http://scihub22266oqcxt.onion/10.1016/j.heares.2015.06.005 C4676957!4676957!26074307     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26074307&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Hear+Res 2016 ; 334 (ä): 20-9 Nephropedia Template TP {{tp|p=26074307|t=2016. Tinnitus: Maladaptive Auditory-Somatosensory Plasticity |pdf=|usr=}}{{26074307}} gab.com Text Tinnitus: Maladaptive Auditory-Somatosensory Plasticity JO: Hear Res http://www.kidney.de/pget.php?&tw=1&pmid=26074307 #FOAvip Tinnitus, the phantom perception of sound, is physiologically characterized by an increase in spontaneous neural activity in the central auditory system. However, as tinnitus is often associated with hearing impairment, it is unclear how a decrease of afferent drive can result in central hyperactivity. In this review, we first assess methods for tinnitus induction and objective measures of the tinnitus percept in animal models. From animal studies, we discuss evidence that tinnitus originates in the cochlear nucleus (CN), and hypothesize mechanisms whereby hyperactivity may develop in the CN after peripheral auditory nerve damage. We elaborate how this process is likely mediated by plasticity of auditory-somatosensory integration in the CN: the circuitry in normal circumstances maintains a balance of auditory and somatosensory activities, and loss of auditory inputs alters the balance of auditory somatosensory integration in a stimulus timing dependent manner, which propels the circuit towards hyperactivity. Understanding the mechanisms underlying tinnitus generation is essential for its prevention and treatment. Twit Text FOAVip Tinnitus: Maladaptive Auditory-Somatosensory Plasticity ????Hear Res http://www.kidney.de/pget.php?&tw=1&pmid=26074307 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26074307 #FOAvip English Wikipedia <ref>{{Cite pmid|26074307}}</ref> Tinnitus: Maladaptive Auditory-Somatosensory Plasticity #MMPMID26074307 Wu C; Stefanescu RA; Martel DT; Shore SE Hear Res 2016[Apr]; 334 (ä): 20-9 PMID26074307show ga Tinnitus, the phantom perception of sound, is physiologically characterized by an increase in spontaneous neural activity in the central auditory system. However, as tinnitus is often associated with hearing impairment, it is unclear how a decrease of afferent drive can result in central hyperactivity. In this review, we first assess methods for tinnitus induction and objective measures of the tinnitus percept in animal models. From animal studies, we discuss evidence that tinnitus originates in the cochlear nucleus (CN), and hypothesize mechanisms whereby hyperactivity may develop in the CN after peripheral auditory nerve damage. We elaborate how this process is likely mediated by plasticity of auditory-somatosensory integration in the CN: the circuitry in normal circumstances maintains a balance of auditory and somatosensory activities, and loss of auditory inputs alters the balance of auditory somatosensory integration in a stimulus timing dependent manner, which propels the circuit towards hyperactivity. Understanding the mechanisms underlying tinnitus generation is essential for its prevention and treatment. äTinnitus: Maladaptive Auditory-Somatosensory Plasticity (epmc).pdf DeepDyve Pubget Overpricing