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10.1155/2015/497387 http://scihub22266oqcxt.onion/10.1155/2015/497387 C4647060!4647060 !26612971     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26612971 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Mediators+Inflamm 2015 ; 2015 (?): 497387 Nephropedia Template TP {{tp|p=26612971 |t=2015. Novel Roles for Chloride Channels, Exchangers, and Regulators in Chronic Inflammatory Airway Diseases |pdf=|usr=}}{{26612971 }} gab.com Text Novel Roles for Chloride Channels, Exchangers, and Regulators in Chronic Inflammatory Airway Diseases JO: Mediators Inflamm http://www.kidney.de/pget.php?&tw=1&pmid=26612971 #FOAvip Chloride transport proteins play critical roles in inflammatory airway diseases, contributing to the detrimental aspects of mucus overproduction, mucus secretion, and airway constriction. However, they also play crucial roles in contributing to the innate immune properties of mucus and mucociliary clearance. In this review, we focus on the emerging novel roles for a chloride channel regulator (CLCA1), a calcium-activated chloride channel (TMEM16A), and two chloride exchangers (SLC26A4/pendrin and SLC26A9) in chronic inflammatory airway diseases. Twit Text FOAVip Novel Roles for Chloride Channels, Exchangers, and Regulators in Chronic Inflammatory Airway Diseases ????Mediators Inflamm http://www.kidney.de/pget.php?&tw=1&pmid=26612971 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26612971 #FOAvip English Wikipedia <ref>{{Cite pmid|26612971 }}</ref> Novel Roles for Chloride Channels, Exchangers, and Regulators in Chronic Inflammatory Airway Diseases #MMPMID26612971 Sala-Rabanal M ; Yurtsever Z ; Berry KN ; Brett TJ Mediators Inflamm 2015[]; 2015 (?): 497387 PMID26612971 show ga Chloride transport proteins play critical roles in inflammatory airway diseases, contributing to the detrimental aspects of mucus overproduction, mucus secretion, and airway constriction. However, they also play crucial roles in contributing to the innate immune properties of mucus and mucociliary clearance. In this review, we focus on the emerging novel roles for a chloride channel regulator (CLCA1), a calcium-activated chloride channel (TMEM16A), and two chloride exchangers (SLC26A4/pendrin and SLC26A9) in chronic inflammatory airway diseases. |Anoctamin-1 [MESH] |Antiporters/*physiology [MESH] |Asthma/*etiology [MESH] |Chloride Channels/*physiology [MESH] |Cystic Fibrosis Transmembrane Conductance Regulator/physiology [MESH] |Humans [MESH] |Membrane Transport Proteins/*physiology [MESH] |Neoplasm Proteins/*physiology [MESH] |Pulmonary Disease, Chronic Obstructive/*etiology [MESH] |STAT6 Transcription Factor/physiology [MESH]Novel Roles for Chloride Channels, Exchangers, and Regulators in Chron(epmc).pdf DeepDyve Pubget Overpricing