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10.1155/2015/634865 http://scihub22266oqcxt.onion/10.1155/2015/634865 C4639645!4639645 !26601108     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26601108 &cmd=llinks): Failed to open stream: HTTP request failed! 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Breast Cancer-Derived Extracellular Vesicles: Characterization and Contribution to the Metastatic Phenotype |pdf=|usr=}}{{26601108 }} gab.com Text Breast Cancer-Derived Extracellular Vesicles: Characterization and Contribution to the Metastatic Phenotype JO: Biomed Res Int http://www.kidney.de/pget.php?&tw=1&pmid=26601108 #FOAvip The study of extracellular vesicles (EVs) in cancer progression is a complex and rapidly evolving field. Whole categories of cellular interactions in cancer which were originally presumed to be due solely to soluble secreted molecules have now evolved to include membrane-enclosed extracellular vesicles (EVs), which include both exosomes and shed microvesicles (MVs), and can contain many of the same molecules as those secreted in soluble form but many different molecules as well. EVs released by cancer cells can transfer mRNA, miRNA, and proteins to different recipient cells within the tumor microenvironment, in both an autocrine and paracrine manner, causing a significant impact on signaling pathways, mRNA transcription, and protein expression. The transfer of EVs to target cells, in turn, supports cancer growth, immunosuppression, and metastasis formation. This review focuses exclusively on breast cancer EVs with an emphasis on breast cancer-derived exosomes, keeping in mind that breast cancer-derived EVs share some common physical properties with EVs of other cancers. Twit Text FOAVip Breast Cancer-Derived Extracellular Vesicles: Characterization and Contribution to the Metastatic Phenotype ????Biomed Res Int http://www.kidney.de/pget.php?&tw=1&pmid=26601108 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26601108 #FOAvip English Wikipedia <ref>{{Cite pmid|26601108 }}</ref> Breast Cancer-Derived Extracellular Vesicles: Characterization and Contribution to the Metastatic Phenotype #MMPMID26601108 Green TM ; Alpaugh ML ; Barsky SH ; Rappa G ; Lorico A Biomed Res Int 2015[]; 2015 (ä): 634865 PMID26601108 show ga The study of extracellular vesicles (EVs) in cancer progression is a complex and rapidly evolving field. Whole categories of cellular interactions in cancer which were originally presumed to be due solely to soluble secreted molecules have now evolved to include membrane-enclosed extracellular vesicles (EVs), which include both exosomes and shed microvesicles (MVs), and can contain many of the same molecules as those secreted in soluble form but many different molecules as well. EVs released by cancer cells can transfer mRNA, miRNA, and proteins to different recipient cells within the tumor microenvironment, in both an autocrine and paracrine manner, causing a significant impact on signaling pathways, mRNA transcription, and protein expression. The transfer of EVs to target cells, in turn, supports cancer growth, immunosuppression, and metastasis formation. This review focuses exclusively on breast cancer EVs with an emphasis on breast cancer-derived exosomes, keeping in mind that breast cancer-derived EVs share some common physical properties with EVs of other cancers. |*Gene Expression Regulation, Neoplastic [MESH] |*Signal Transduction [MESH] |Breast Neoplasms/*metabolism/pathology [MESH] |Cell-Derived Microparticles/*metabolism/pathology [MESH] |Exosomes/*metabolism/pathology [MESH] |Female [MESH] |Humans [MESH] |MicroRNAs/metabolism [MESH] |Neoplasm Metastasis [MESH] |Neoplasm Proteins/metabolism [MESH] |RNA, Neoplasm/metabolism [MESH] |RNA, Transfer/metabolism [MESH]Breast Cancer-Derived Extracellular Vesicles: Characterization and Con(epmc).pdf DeepDyve Pubget Overpricing