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10.1099/vir.0.000107 http://scihub22266oqcxt.onion/10.1099/vir.0.000107 C4635457!4635457!25724670     free     free     free Deprecated: Implicit conversion from float 229.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 229.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Gen+Virol 2015 ; 96 (Pt 7): 1890-8 Nephropedia Template TP {{tp|p=25724670|t=2015. Analysis of the history and spread of HIV-1 in Uganda using phylodynamics |pdf=|usr=}}{{25724670}} gab.com Text Analysis of the history and spread of HIV-1 in Uganda using phylodynamics JO: J Gen Virol http://www.kidney.de/pget.php?&tw=1&pmid=25724670 #FOAvip HIV prevalence has decreased in Uganda since the 1990s, but remains substantial within high-risk groups. Here, we reconstruct the history and spread of HIV subtypes A1 and D in Uganda and explore the transmission dynamics in high-risk populations. We analysed HIV pol sequences from female sex workers in Kampala (n?=?42), Lake Victoria fisher-folk (n?=?46) and a rural clinical cohort (n?=?74), together with publicly available sequences from adjacent regions in Uganda (n?=?412) and newly generated sequences from samples taken in Kampala in 1986 (n?=?12). Of the sequences from the three Ugandan populations, 60 (37.1?%) were classified as subtype D, 54 (33.3?%) as subtype A1, 31 (19.1?%) as A1/D recombinants, six (3.7?%) as subtype C, one (0.6?%) as subtype G and 10 (6.2?%) as other recombinants. Among the A1/D recombinants we identified a new candidate circulating recombinant form. Phylodynamic and phylogeographic analyses using BEAST indicated that the Ugandan epidemics originated in 1960 (1950?1968) for subtype A1 and 1973 (1970?1977) for D, in rural south-western Uganda with subsequent spread to Kampala. They also showed extensive interconnection with adjacent countries. The sequence analysis shows both epidemics grew exponentially during the 1970s?1980s and decreased from 1992, which agrees with HIV prevalence reports in Uganda. Inclusion of sequences from the 1980s indicated the origin of both epidemics was more recent than expected and substantially narrowed the confidence intervals in comparison to previous estimates. We identified three transmission clusters and ten pairs, none of them including patients from different populations, suggesting active transmission within a structured transmission network. Twit Text FOAVip Analysis of the history and spread of HIV-1 in Uganda using phylodynamics ????J Gen Virol http://www.kidney.de/pget.php?&tw=1&pmid=25724670 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25724670 #FOAvip English Wikipedia <ref>{{Cite pmid|25724670}}</ref> Analysis of the history and spread of HIV-1 in Uganda using phylodynamics #MMPMID25724670 Yebra G; Ragonnet-Cronin M; Ssemwanga D; Parry CM; Logue CH; Cane PA; Kaleebu P; Brown AJL J Gen Virol 2015[Jul]; 96 (Pt 7): 1890-8 PMID25724670show ga HIV prevalence has decreased in Uganda since the 1990s, but remains substantial within high-risk groups. Here, we reconstruct the history and spread of HIV subtypes A1 and D in Uganda and explore the transmission dynamics in high-risk populations. We analysed HIV pol sequences from female sex workers in Kampala (n?=?42), Lake Victoria fisher-folk (n?=?46) and a rural clinical cohort (n?=?74), together with publicly available sequences from adjacent regions in Uganda (n?=?412) and newly generated sequences from samples taken in Kampala in 1986 (n?=?12). Of the sequences from the three Ugandan populations, 60 (37.1?%) were classified as subtype D, 54 (33.3?%) as subtype A1, 31 (19.1?%) as A1/D recombinants, six (3.7?%) as subtype C, one (0.6?%) as subtype G and 10 (6.2?%) as other recombinants. Among the A1/D recombinants we identified a new candidate circulating recombinant form. Phylodynamic and phylogeographic analyses using BEAST indicated that the Ugandan epidemics originated in 1960 (1950?1968) for subtype A1 and 1973 (1970?1977) for D, in rural south-western Uganda with subsequent spread to Kampala. They also showed extensive interconnection with adjacent countries. The sequence analysis shows both epidemics grew exponentially during the 1970s?1980s and decreased from 1992, which agrees with HIV prevalence reports in Uganda. Inclusion of sequences from the 1980s indicated the origin of both epidemics was more recent than expected and substantially narrowed the confidence intervals in comparison to previous estimates. We identified three transmission clusters and ten pairs, none of them including patients from different populations, suggesting active transmission within a structured transmission network. äAnalysis of the history and spread of HIV-1 in Uganda using phylodynam(epmc).pdf DeepDyve Pubget Overpricing