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10.1210/me.2015-1206 http://scihub22266oqcxt.onion/10.1210/me.2015-1206 C4627606!4627606!26393296     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Mol+Endocrinol 2015 ; 29 (11): 1535-48 Nephropedia Template TP {{tp|p=26393296|t=2015. Minireview: Emerging Roles for Extracellular Vesicles in Diabetes and Related Metabolic Disorders |pdf=|usr=}}{{26393296}} gab.com Text Minireview: Emerging Roles for Extracellular Vesicles in Diabetes and Related Metabolic Disorders JO: Mol Endocrinol http://www.kidney.de/pget.php?&tw=1&pmid=26393296 #FOAvip Extracellular vesicles (EVs), membrane-contained vesicles released by most cell types, have attracted a large amount of research interest over the past decade. Because of their ability to transfer cargo via regulated processes, causing functional impacts on recipient cells, these structures may play important roles in cell-cell communication and have implications in the physiology of numerous organ systems. In addition, EVs have been described in most human biofluids and have wide potential as relatively noninvasive biomarkers of various pathologic conditions. Specifically, EVs produced by the pancreatic ?-cell have been demonstrated to regulate physiologic and pathologic responses to ?-cell stress, including ?-cell proliferation and apoptosis. ?-Cell EVs are also capable of interacting with immune cells and may contribute to the activation of autoimmune processes that trigger or propagate ?-cell inflammation and destruction during the development of diabetes. EVs from adipose tissue have been shown to contribute to the development of the chronic inflammation and insulin resistance associated with obesity and metabolic syndrome via interactions with other adipose, liver, and muscle cells. Circulating EVs may also serve as biomarkers for metabolic derangements and complications associated with diabetes. This minireview describes the properties of EVs in general, followed by a more focused review of the literature describing EVs affecting the ?-cell, ?-cell autoimmunity, and the development of insulin resistance, which all have the potential to affect development of type 1 or type 2 diabetes. Twit Text FOAVip Minireview: Emerging Roles for Extracellular Vesicles in Diabetes and Related Metabolic Disorders ????Mol Endocrinol http://www.kidney.de/pget.php?&tw=1&pmid=26393296 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26393296 #FOAvip English Wikipedia <ref>{{Cite pmid|26393296}}</ref> Minireview: Emerging Roles for Extracellular Vesicles in Diabetes and Related Metabolic Disorders #MMPMID26393296 Lakhter AJ; Sims EK Mol Endocrinol 2015[Nov]; 29 (11): 1535-48 PMID26393296show ga Extracellular vesicles (EVs), membrane-contained vesicles released by most cell types, have attracted a large amount of research interest over the past decade. Because of their ability to transfer cargo via regulated processes, causing functional impacts on recipient cells, these structures may play important roles in cell-cell communication and have implications in the physiology of numerous organ systems. In addition, EVs have been described in most human biofluids and have wide potential as relatively noninvasive biomarkers of various pathologic conditions. Specifically, EVs produced by the pancreatic ?-cell have been demonstrated to regulate physiologic and pathologic responses to ?-cell stress, including ?-cell proliferation and apoptosis. ?-Cell EVs are also capable of interacting with immune cells and may contribute to the activation of autoimmune processes that trigger or propagate ?-cell inflammation and destruction during the development of diabetes. EVs from adipose tissue have been shown to contribute to the development of the chronic inflammation and insulin resistance associated with obesity and metabolic syndrome via interactions with other adipose, liver, and muscle cells. Circulating EVs may also serve as biomarkers for metabolic derangements and complications associated with diabetes. This minireview describes the properties of EVs in general, followed by a more focused review of the literature describing EVs affecting the ?-cell, ?-cell autoimmunity, and the development of insulin resistance, which all have the potential to affect development of type 1 or type 2 diabetes. äMinireview: Emerging Roles for Extracellular Vesicles in Diabetes and (epmc).pdf DeepDyve Pubget Overpricing