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10.1007/s00281-015-0515-3 http://scihub22266oqcxt.onion/10.1007/s00281-015-0515-3 C4618768!4618768!26223505     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Semin+Immunopathol 2015 ; 37 (6): 625-38 Nephropedia Template TP {{tp|p=26223505|t=2015. Control of autoimmune CNS inflammation by astrocytes |pdf=|usr=}}{{26223505}} gab.com Text Control of autoimmune CNS inflammation by astrocytes JO: Semin Immunopathol http://www.kidney.de/pget.php?&tw=1&pmid=26223505 #FOAvip Multiple Sclerosis is a neurologic disease caused by immune cell infiltration into the central nervous system, resulting in grey and white matter inflammation, progressive demyelination and neuronal loss. Astrocytes, the most abundant cell population in the CNS, have been considered inert scaffold- or housekeeping cells for many years. However, recently it has become clear that this cell population actively modulates the immune response in the CNS at multiple levels. While being exposed to a plethora of cytokines during ongoing autoimmune inflammation, astrocytes modulate local CNS inflammation by secreting cytokines and chemokines, among other factors. This review article gives an overview of the most recent understanding about cytokine networks operational in astrocytes during autoimmune neuroinflammation and highlights potential targets for immunomodulatory therapies for Multiple Sclerosis. Twit Text FOAVip Control of autoimmune CNS inflammation by astrocytes ????Semin Immunopathol http://www.kidney.de/pget.php?&tw=1&pmid=26223505 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26223505 #FOAvip English Wikipedia <ref>{{Cite pmid|26223505}}</ref> Control of autoimmune CNS inflammation by astrocytes #MMPMID26223505 Rothhammer V; Quintana FJ Semin Immunopathol 2015[Nov]; 37 (6): 625-38 PMID26223505show ga Multiple Sclerosis is a neurologic disease caused by immune cell infiltration into the central nervous system, resulting in grey and white matter inflammation, progressive demyelination and neuronal loss. Astrocytes, the most abundant cell population in the CNS, have been considered inert scaffold- or housekeeping cells for many years. However, recently it has become clear that this cell population actively modulates the immune response in the CNS at multiple levels. While being exposed to a plethora of cytokines during ongoing autoimmune inflammation, astrocytes modulate local CNS inflammation by secreting cytokines and chemokines, among other factors. This review article gives an overview of the most recent understanding about cytokine networks operational in astrocytes during autoimmune neuroinflammation and highlights potential targets for immunomodulatory therapies for Multiple Sclerosis. äControl of autoimmune CNS inflammation by astrocytes (epmc).pdf DeepDyve Pubget Overpricing