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10.1002/stem.1566 http://scihub22266oqcxt.onion/10.1002/stem.1566 C4603545!4603545 !24123696     free     free     free Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\24123696 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Stem+Cells 2014 ; 32 (3 ): 822-7 Nephropedia Template TP {{tp|p=24123696 |t=2014. Brief report: Dclk1 deletion in tuft cells results in impaired epithelial repair after radiation injury |pdf=|usr=}}{{24123696 }} gab.com Text Brief report: Dclk1 deletion in tuft cells results in impaired epithelial repair after radiation injury JO: Stem Cells http://www.kidney.de/pget.php?&tw=1&pmid=24123696 #FOAvip The role of Dclk1(+) tuft cells in the replacement of intestinal epithelia and reestablishing the epithelial barrier after severe genotoxic insult is completely unknown. Successful restoration requires precise coordination between the cells within each crypt subunit. While the mechanisms that control this response remain largely uncertain, the radiation model remains an exceptional surrogate for stem cell-associated crypt loss. Following the creation of Dclk1-intestinal-epithelial-deficient Villin-Cre;Dclk1(flox/flox) mice, widespread gene expression changes were detected in isolated intestinal epithelia during homeostasis. While the number of surviving crypts was unaffected, Villin-Cre;Dclk1(flox/flox) mice failed to maintain tight junctions and died at approximately 5 days, where Dclk1(flox/flox) mice lived until day 10 following radiation injury. These findings suggest that Dclk1 plays a functional role critical in the epithelial restorative response. Twit Text FOAVip Brief report: Dclk1 deletion in tuft cells results in impaired epithelial repair after radiation injury ????Stem Cells http://www.kidney.de/pget.php?&tw=1&pmid=24123696 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24123696 #FOAvip English Wikipedia <ref>{{Cite pmid|24123696 }}</ref> Brief report: Dclk1 deletion in tuft cells results in impaired epithelial repair after radiation injury #MMPMID24123696 May R ; Qu D ; Weygant N ; Chandrakesan P ; Ali N ; Lightfoot SA ; Li L ; Sureban SM ; Houchen CW Stem Cells 2014[Mar]; 32 (3 ): 822-7 PMID24123696 show ga The role of Dclk1(+) tuft cells in the replacement of intestinal epithelia and reestablishing the epithelial barrier after severe genotoxic insult is completely unknown. Successful restoration requires precise coordination between the cells within each crypt subunit. While the mechanisms that control this response remain largely uncertain, the radiation model remains an exceptional surrogate for stem cell-associated crypt loss. Following the creation of Dclk1-intestinal-epithelial-deficient Villin-Cre;Dclk1(flox/flox) mice, widespread gene expression changes were detected in isolated intestinal epithelia during homeostasis. While the number of surviving crypts was unaffected, Villin-Cre;Dclk1(flox/flox) mice failed to maintain tight junctions and died at approximately 5 days, where Dclk1(flox/flox) mice lived until day 10 following radiation injury. These findings suggest that Dclk1 plays a functional role critical in the epithelial restorative response. |*Gene Deletion [MESH] |*Wound Healing [MESH] |Animals [MESH] |Doublecortin-Like Kinases [MESH] |Epithelial Cells/*metabolism/*pathology [MESH] |Gene Expression Regulation [MESH] |Intestinal Mucosa/*pathology [MESH] |Mice [MESH] |Protein Serine-Threonine Kinases/*genetics [MESH] |RNA, Messenger/genetics/metabolism [MESH] |Radiation Injuries/metabolism/*pathology [MESH] |Receptors, Notch/genetics/metabolism [MESH] |Stem Cells/cytology/metabolism [MESH] |Survival Analysis [MESH]Brief report: Dclk1 deletion in tuft cells results in impaired epithel(epmc).pdf DeepDyve Pubget Overpricing