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10.1155/2015/795602 http://scihub22266oqcxt.onion/10.1155/2015/795602 C4602327!4602327!26491536     free     free     free Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Oxid+Med+Cell+Longev 2015 ; 2015 (ä): ä Nephropedia Template TP {{tp|p=26491536|t=2015. Chemotherapy-Induced Cardiotoxicity: Overview of the Roles of Oxidative Stress |pdf=|usr=}}{{26491536}} gab.com Text Chemotherapy-Induced Cardiotoxicity: Overview of the Roles of Oxidative Stress JO: Oxid Med Cell Longev http://www.kidney.de/pget.php?&tw=1&pmid=26491536 #FOAvip Chemotherapy-induced cardiotoxicity is a serious complication that poses a serious threat to life and limits the clinical use of various chemotherapeutic agents, particularly the anthracyclines. Understanding molecular mechanisms of chemotherapy-induced cardiotoxicity is a key to effective preventive strategies and improved chemotherapy regimen. Although no reliable and effective preventive treatment has become available, numerous evidence demonstrates that chemotherapy-induced cardiotoxicity involves the generation of reactive oxygen species (ROS). This review provides an overview of the roles of oxidative stress in chemotherapy-induced cardiotoxicity using doxorubicin, which is one of the most effective chemotherapeutic agents against a wide range of cancers, as an example. Current understanding in the molecular mechanisms of ROS-mediated cardiotoxicity will be explored and discussed, with emphasis on cardiomyocyte apoptosis leading to cardiomyopathy. The review will conclude with perspectives on model development needed to facilitate further progress and understanding on chemotherapy-induced cardiotoxicity. Twit Text FOAVip Chemotherapy-Induced Cardiotoxicity: Overview of the Roles of Oxidative Stress ????Oxid Med Cell Longev http://www.kidney.de/pget.php?&tw=1&pmid=26491536 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26491536 #FOAvip English Wikipedia <ref>{{Cite pmid|26491536}}</ref> Chemotherapy-Induced Cardiotoxicity: Overview of the Roles of Oxidative Stress #MMPMID26491536 Angsutararux P; Luanpitpong S; Issaragrisil S Oxid Med Cell Longev 2015[]; 2015 (ä): ä PMID26491536show ga Chemotherapy-induced cardiotoxicity is a serious complication that poses a serious threat to life and limits the clinical use of various chemotherapeutic agents, particularly the anthracyclines. Understanding molecular mechanisms of chemotherapy-induced cardiotoxicity is a key to effective preventive strategies and improved chemotherapy regimen. Although no reliable and effective preventive treatment has become available, numerous evidence demonstrates that chemotherapy-induced cardiotoxicity involves the generation of reactive oxygen species (ROS). This review provides an overview of the roles of oxidative stress in chemotherapy-induced cardiotoxicity using doxorubicin, which is one of the most effective chemotherapeutic agents against a wide range of cancers, as an example. Current understanding in the molecular mechanisms of ROS-mediated cardiotoxicity will be explored and discussed, with emphasis on cardiomyocyte apoptosis leading to cardiomyopathy. The review will conclude with perspectives on model development needed to facilitate further progress and understanding on chemotherapy-induced cardiotoxicity. äChemotherapy-Induced Cardiotoxicity: Overview of the Roles of Oxidativ(epmc).pdf DeepDyve Pubget Overpricing