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10.3390/jfb6030963 http://scihub22266oqcxt.onion/10.3390/jfb6030963 C4598687!4598687!26402712     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Funct+Biomater 2015 ; 6 (3): 963-85 Nephropedia Template TP {{tp|p=26402712|t=2015. A Closer Look at Schlemm?s Canal Cell Physiology: Implications for Biomimetics |pdf=|usr=}}{{26402712}} gab.com Text A Closer Look at Schlemm s Canal Cell Physiology: Implications for Biomimetics JO: J Funct Biomater http://www.kidney.de/pget.php?&tw=1&pmid=26402712 #FOAvip Among ocular pathologies, glaucoma is the second leading cause of progressive vision loss, expected to affect 80 million people worldwide by 2020. A primary cause of glaucoma appears to be damage to the conventional outflow tract. Conventional outflow tissues, a composite of the trabecular meshwork and the Schlemm?s canal, regulate and maintain homeostatic responses to intraocular pressure. In glaucoma, filtration of aqueous humor into the Schlemm?s canal is hindered, leading to an increase in intraocular pressure and subsequent damage to the optic nerve, with progressive vision loss. The Schlemm?s canal encompasses a unique endothelium. Recent advances in culturing and manipulating Schlemm?s canal cells have elucidated several aspects of their physiology, including ultrastructure, cell-specific marker expression, and biomechanical properties. This review highlights these advances and discusses implications for engineering a 3D, biomimetic, in vitro model of the Schlemm?s canal endothelium to further advance glaucoma research, including drug testing and gene therapy screening. Twit Text FOAVip A Closer Look at Schlemm s Canal Cell Physiology: Implications for Biomimetics ????J Funct Biomater http://www.kidney.de/pget.php?&tw=1&pmid=26402712 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26402712 #FOAvip English Wikipedia <ref>{{Cite pmid|26402712}}</ref> A Closer Look at Schlemm?s Canal Cell Physiology: Implications for Biomimetics #MMPMID26402712 Dautriche CN; Tian Y; Xie Y; Sharfstein ST J Funct Biomater 2015[Sep]; 6 (3): 963-85 PMID26402712show ga Among ocular pathologies, glaucoma is the second leading cause of progressive vision loss, expected to affect 80 million people worldwide by 2020. A primary cause of glaucoma appears to be damage to the conventional outflow tract. Conventional outflow tissues, a composite of the trabecular meshwork and the Schlemm?s canal, regulate and maintain homeostatic responses to intraocular pressure. In glaucoma, filtration of aqueous humor into the Schlemm?s canal is hindered, leading to an increase in intraocular pressure and subsequent damage to the optic nerve, with progressive vision loss. The Schlemm?s canal encompasses a unique endothelium. Recent advances in culturing and manipulating Schlemm?s canal cells have elucidated several aspects of their physiology, including ultrastructure, cell-specific marker expression, and biomechanical properties. This review highlights these advances and discusses implications for engineering a 3D, biomimetic, in vitro model of the Schlemm?s canal endothelium to further advance glaucoma research, including drug testing and gene therapy screening. äA Closer Look at Schlemm?s Canal Cell Physiology: Implications for Bio(epmc).pdf DeepDyve Pubget Overpricing