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10.4137/BIC.S29329 http://scihub22266oqcxt.onion/10.4137/BIC.S29329 C4597444!4597444!26483612     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Biomark+Cancer 2015 ; 7 (Suppl 2): 19-24 Nephropedia Template TP {{tp|p=26483612|t=2015. Ameloblastoma: A Review of Recent Molecular Pathogenetic Discoveries |pdf=|usr=}}{{26483612}} gab.com Text Ameloblastoma: A Review of Recent Molecular Pathogenetic Discoveries JO: Biomark Cancer http://www.kidney.de/pget.php?&tw=1&pmid=26483612 #FOAvip Ameloblastoma is an odontogenic neoplasm whose molecular pathogenesis has only recently been elucidated. The discovery of recurrent activating mutations in FGFR2, BRAF, and RAS in a large majority of ameloblastomas has implicated dysregulation of MAPK pathway signaling as a critical step in the pathogenesis of this tumor. Some degree of controversy exists regarding the role of mutations affecting the sonic hedgehog (SHH) pathway, specifically Smoothened (SMO), which have been postulated to serve as either an alternative pathogenetic mechanism or secondary mutations. Here, we review recent advances in our understanding of the molecular pathogenesis of ameloblastoma as well as the diagnostic, prognostic, and therapeutic implications of these discoveries. Twit Text FOAVip Ameloblastoma: A Review of Recent Molecular Pathogenetic Discoveries ????Biomark Cancer http://www.kidney.de/pget.php?&tw=1&pmid=26483612 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26483612 #FOAvip English Wikipedia <ref>{{Cite pmid|26483612}}</ref> Ameloblastoma: A Review of Recent Molecular Pathogenetic Discoveries #MMPMID26483612 Brown NA; Betz BL Biomark Cancer 2015[]; 7 (Suppl 2): 19-24 PMID26483612show ga Ameloblastoma is an odontogenic neoplasm whose molecular pathogenesis has only recently been elucidated. The discovery of recurrent activating mutations in FGFR2, BRAF, and RAS in a large majority of ameloblastomas has implicated dysregulation of MAPK pathway signaling as a critical step in the pathogenesis of this tumor. Some degree of controversy exists regarding the role of mutations affecting the sonic hedgehog (SHH) pathway, specifically Smoothened (SMO), which have been postulated to serve as either an alternative pathogenetic mechanism or secondary mutations. Here, we review recent advances in our understanding of the molecular pathogenesis of ameloblastoma as well as the diagnostic, prognostic, and therapeutic implications of these discoveries. äAmeloblastoma: A Review of Recent Molecular Pathogenetic Discoveries (epmc).pdf DeepDyve Pubget Overpricing