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10.4103/1673-5374.162759 http://scihub22266oqcxt.onion/10.4103/1673-5374.162759 C4590239!4590239 !26487854     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26487854 &cmd=llinks): Failed to open stream: HTTP request failed! 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Bone marrow mesenchymal stem cells transplantation promotes the release of endogenous erythropoietin after ischemic stroke |pdf=|usr=}}{{26487854 }} gab.com Text Bone marrow mesenchymal stem cells transplantation promotes the release of endogenous erythropoietin after ischemic stroke JO: Neural Regen Res http://www.kidney.de/pget.php?&tw=1&pmid=26487854 #FOAvip This study investigated whether bone marrow mesenchymal stem cell (BMSC) transplantation protected ischemic cerebral injury by stimulating endogenous erythropoietin. The model of ischemic stroke was established in rats through transient middle cerebral artery occlusion. Twenty-four hours later, 1 × 10(6) human BMSCs (hBMSCs) were injected into the tail vein. Fourteen days later, we found that hBMSCs promoted the release of endogenous erythropoietin in the ischemic region of rats. Simultaneously, 3 ?g/d soluble erythropoietin receptor (sEPOR) was injected into the lateral ventricle, and on the next 13 consecutive days. sEPOR blocked the release of endogenous erythropoietin. The neurogenesis in the subventricular zone was less in the hBMSCs + sEPOR group than in the hBMSCs + heat-denatured sEPOR group. The adhesive-removal test result and the modified Neurological Severity Scores (mNSS) were lower in the hBMSCs + sEPOR group than in the heat-denatured sEPOR group. The adhesive-removal test result and mNSS were similar between the hBMSCs + heat-denatured sEPOR group and the hBMSCs + sEPOR group. These findings confirm that BMSCs contribute to neurogenesis and improve neurological function by promoting the release of endogenous erythropoietin following ischemic stroke. Twit Text FOAVip Bone marrow mesenchymal stem cells transplantation promotes the release of endogenous erythropoietin after ischemic stroke ????Neural Regen Res http://www.kidney.de/pget.php?&tw=1&pmid=26487854 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26487854 #FOAvip English Wikipedia <ref>{{Cite pmid|26487854 }}</ref> Bone marrow mesenchymal stem cells transplantation promotes the release of endogenous erythropoietin after ischemic stroke #MMPMID26487854 Lv W ; Li WY ; Xu XY ; Jiang H ; Bang OY Neural Regen Res 2015[Aug]; 10 (8 ): 1265-70 PMID26487854 show ga This study investigated whether bone marrow mesenchymal stem cell (BMSC) transplantation protected ischemic cerebral injury by stimulating endogenous erythropoietin. The model of ischemic stroke was established in rats through transient middle cerebral artery occlusion. Twenty-four hours later, 1 × 10(6) human BMSCs (hBMSCs) were injected into the tail vein. Fourteen days later, we found that hBMSCs promoted the release of endogenous erythropoietin in the ischemic region of rats. Simultaneously, 3 ?g/d soluble erythropoietin receptor (sEPOR) was injected into the lateral ventricle, and on the next 13 consecutive days. sEPOR blocked the release of endogenous erythropoietin. The neurogenesis in the subventricular zone was less in the hBMSCs + sEPOR group than in the hBMSCs + heat-denatured sEPOR group. The adhesive-removal test result and the modified Neurological Severity Scores (mNSS) were lower in the hBMSCs + sEPOR group than in the heat-denatured sEPOR group. The adhesive-removal test result and mNSS were similar between the hBMSCs + heat-denatured sEPOR group and the hBMSCs + sEPOR group. These findings confirm that BMSCs contribute to neurogenesis and improve neurological function by promoting the release of endogenous erythropoietin following ischemic stroke. äBone marrow mesenchymal stem cells transplantation promotes the releas(epmc).pdf DeepDyve Pubget Overpricing