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10.1080/19381980.2015.1010983 http://scihub22266oqcxt.onion/10.1080/19381980.2015.1010983 C4579972!4579972 !26413189     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26413189 &cmd=llinks): Failed to open stream: HTTP request failed! 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A vitamin D analog inhibits Th2 cytokine- and TGF? -induced periostin production in fibroblasts: a potential role for vitamin D in skin sclerosis |pdf=|usr=}}{{26413189 }} gab.com Text A vitamin D analog inhibits Th2 cytokine- and TGF -induced periostin production in fibroblasts: a potential role for vitamin D in skin sclerosis JO: Dermatoendocrinol http://www.kidney.de/pget.php?&tw=1&pmid=26413189 #FOAvip Scleroderma is an autoimmune disease characterized by extracellular matrix deposition and inflammation. Topical vitamin D analogs have been reported as effective treatments for scleroderma. We previously reported that a matricellular protein, periostin (POSTN), contributes to pathogenesis of scleroderma as POSTN knockout mice were resistant to bleomycin (BLM)-induced scleroderma. We investigated whether a vitamin D analog affects the expression of POSTN in dermal fibroblasts and in a BLM-induced scleroderma model. The vitamin D analog, maxacalcitol (22-oxacalcitriol [OCT]), was applied to dermal fibroblasts and POSTN expression was measured. The effect of OCT on Th2 cytokine- and TGF?-induced POTSN and Collagen 1 ? 1 (Col1A1) expression was also assessed. In vivo, OCT was administered to BLM-induced scleroderma model and outcomes were determined by dermal thickness, collagen density and POSTN expression. Treatment with OCT significantly decreased POSTN expression in dermal fibroblasts. Th2 cytokine- and TGF?-induced expression of POSTN and Col1A1 was also suppressed by OCT. In vivo, OCT administration decreased the density of collagen bundles and POSTN expression in a BLM-induced scleroderma model. In addition to the previously reported immunosuppressive effect, the vitamin D analog OCT might be effective to treat scleroderma, in part through inhibition of Th2 cytokine- and TGF?-induced POSTN expression. Twit Text FOAVip A vitamin D analog inhibits Th2 cytokine- and TGF -induced periostin production in fibroblasts: a potential role for vitamin D in skin sclerosis ????Dermatoendocrinol http://www.kidney.de/pget.php?&tw=1&pmid=26413189 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26413189 #FOAvip English Wikipedia <ref>{{Cite pmid|26413189 }}</ref> A vitamin D analog inhibits Th2 cytokine- and TGF? -induced periostin production in fibroblasts: a potential role for vitamin D in skin sclerosis #MMPMID26413189 Terao M ; Yang L ; Matsumura S ; Yutani M ; Murota H ; Katayama I Dermatoendocrinol 2015[Jan]; 7 (1 ): e1010983 PMID26413189 show ga Scleroderma is an autoimmune disease characterized by extracellular matrix deposition and inflammation. Topical vitamin D analogs have been reported as effective treatments for scleroderma. We previously reported that a matricellular protein, periostin (POSTN), contributes to pathogenesis of scleroderma as POSTN knockout mice were resistant to bleomycin (BLM)-induced scleroderma. We investigated whether a vitamin D analog affects the expression of POSTN in dermal fibroblasts and in a BLM-induced scleroderma model. The vitamin D analog, maxacalcitol (22-oxacalcitriol [OCT]), was applied to dermal fibroblasts and POSTN expression was measured. The effect of OCT on Th2 cytokine- and TGF?-induced POTSN and Collagen 1 ? 1 (Col1A1) expression was also assessed. In vivo, OCT was administered to BLM-induced scleroderma model and outcomes were determined by dermal thickness, collagen density and POSTN expression. Treatment with OCT significantly decreased POSTN expression in dermal fibroblasts. Th2 cytokine- and TGF?-induced expression of POSTN and Col1A1 was also suppressed by OCT. In vivo, OCT administration decreased the density of collagen bundles and POSTN expression in a BLM-induced scleroderma model. In addition to the previously reported immunosuppressive effect, the vitamin D analog OCT might be effective to treat scleroderma, in part through inhibition of Th2 cytokine- and TGF?-induced POSTN expression. äA vitamin D analog inhibits Th2 cytokine- and TGF? -induced periostin (epmc).pdf DeepDyve Pubget Overpricing