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10.1016/j.pneurobio.2015.02.003 http://scihub22266oqcxt.onion/10.1016/j.pneurobio.2015.02.003 C4578232!4578232 !25802011     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\25802011 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Prog+Neurobiol 2015 ; 127-128 (ä): 1-22 Nephropedia Template TP {{tp|p=25802011 |t=2015. The role of immune cells, glia and neurons in white and gray matter pathology in multiple sclerosis |pdf=|usr=}}{{25802011 }} gab.com Text The role of immune cells, glia and neurons in white and gray matter pathology in multiple sclerosis JO: Prog Neurobiol http://www.kidney.de/pget.php?&tw=1&pmid=25802011 #FOAvip Multiple sclerosis is one of the most common causes of chronic neurological disability beginning in early to middle adult life. Multiple sclerosis is idiopathic in nature, yet increasing correlative evidence supports a strong association between one's genetic predisposition, the environment and the immune system. Symptoms of multiple sclerosis have primarily been shown to result from a disruption in the integrity of myelinated tracts within the white matter of the central nervous system. However, recent research has also highlighted the hitherto underappreciated involvement of gray matter in multiple sclerosis disease pathophysiology, which may be especially relevant when considering the accumulation of irreversible damage and progressive disability. This review aims at providing a comprehensive overview of the interplay between inflammation, glial/neuronal damage and regeneration throughout the course of multiple sclerosis via the analysis of both white and gray matter lesional pathology. Further, we describe the common pathological mechanisms underlying both relapsing and progressive forms of multiple sclerosis, and analyze how current (as well as future) treatments may interact and/or interfere with its pathology. Understanding the putative mechanisms that drive disease pathogenesis will be key in helping to develop effective therapeutic strategies to prevent, mitigate, and treat the diverse morbidities associated with multiple sclerosis. Twit Text FOAVip The role of immune cells, glia and neurons in white and gray matter pathology in multiple sclerosis ????Prog Neurobiol http://www.kidney.de/pget.php?&tw=1&pmid=25802011 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25802011 #FOAvip English Wikipedia <ref>{{Cite pmid|25802011 }}</ref> The role of immune cells, glia and neurons in white and gray matter pathology in multiple sclerosis #MMPMID25802011 Mallucci G ; Peruzzotti-Jametti L ; Bernstock JD ; Pluchino S Prog Neurobiol 2015[Apr]; 127-128 (ä): 1-22 PMID25802011 show ga Multiple sclerosis is one of the most common causes of chronic neurological disability beginning in early to middle adult life. Multiple sclerosis is idiopathic in nature, yet increasing correlative evidence supports a strong association between one's genetic predisposition, the environment and the immune system. Symptoms of multiple sclerosis have primarily been shown to result from a disruption in the integrity of myelinated tracts within the white matter of the central nervous system. However, recent research has also highlighted the hitherto underappreciated involvement of gray matter in multiple sclerosis disease pathophysiology, which may be especially relevant when considering the accumulation of irreversible damage and progressive disability. This review aims at providing a comprehensive overview of the interplay between inflammation, glial/neuronal damage and regeneration throughout the course of multiple sclerosis via the analysis of both white and gray matter lesional pathology. Further, we describe the common pathological mechanisms underlying both relapsing and progressive forms of multiple sclerosis, and analyze how current (as well as future) treatments may interact and/or interfere with its pathology. Understanding the putative mechanisms that drive disease pathogenesis will be key in helping to develop effective therapeutic strategies to prevent, mitigate, and treat the diverse morbidities associated with multiple sclerosis. |Animals [MESH] |Brain/immunology/*pathology [MESH] |Gray Matter/immunology/*pathology [MESH] |Humans [MESH] |Multiple Sclerosis/*pathology/physiopathology [MESH] |Neuroglia/immunology/*pathology [MESH] |Neuroimmunomodulation/physiology [MESH]The role of immune cells, glia and neurons in white and gray matter pa(epmc).pdf DeepDyve Pubget Overpricing