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10.1016/j.tem.2015.07.004

http://scihub22266oqcxt.onion/10.1016/j.tem.2015.07.004
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C4558202!4558202!26239304
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suck abstract from ncbi

pmid26239304      Trends+Endocrinol+Metab 2015 ; 26 (9): 451-2
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  • NETs and traps delay wound healing in diabetes #MMPMID26239304
  • Roth Flach RJ; Czech MP
  • Trends Endocrinol Metab 2015[Sep]; 26 (9): 451-2 PMID26239304show ga
  • Diabetes is associated with dire peripheral sequelae including foot ulcers and amputations. A recent article by Wong et al [2] demystifies this connection by demonstrating that the neutrophil defense mechanism of extruding decondensed chromatin, termed NETosis, mediates delayed wound healing in diabetes and provides a therapeutic strategy for this indication.
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