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10.3402/jom.v7.28788 http://scihub22266oqcxt.onion/10.3402/jom.v7.28788 C4557090!4557090!26329158     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Oral+Microbiol 2015 ; 7 (ä): ä Nephropedia Template TP {{tp|p=26329158|t=2015. Invasion of Porphyromonas gingivalis strains into vascular cells and tissue |pdf=|usr=}}{{26329158}} gab.com Text Invasion of Porphyromonas gingivalis strains into vascular cells and tissue JO: J Oral Microbiol http://www.kidney.de/pget.php?&tw=1&pmid=26329158 #FOAvip Porphyromonas gingivalis is considered a major pathogen in adult periodontitis and is also associated with multiple systemic diseases, for example, cardiovascular diseases. One of its most important virulence factors is invasion of host cells. The invasion process includes attachment, entry/internalization, trafficking, persistence, and exit. The present review discusses these processes related to P. gingivalis in cardiovascular cells and tissue. Although most P. gingivalis strains invade, the invasion capacity of strains and the mechanisms of invasion including intracellular trafficking among them differ. This is consistent with the fact that there are significant differences in the pathogenicity of P. gingivalis strains. P. gingivalis invasion mechanisms are also dependent on types of host cells. Although much is known about the invasion process of P. gingivalis, we still have little knowledge of its exit mechanisms. Nevertheless, it is intriguing that P. gingivalis can remain viable in human cardiovascular cells and atherosclerotic plaque and later exit and re-enter previously uninfected host cells. Twit Text FOAVip Invasion of Porphyromonas gingivalis strains into vascular cells and tissue ????J Oral Microbiol http://www.kidney.de/pget.php?&tw=1&pmid=26329158 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26329158 #FOAvip English Wikipedia <ref>{{Cite pmid|26329158}}</ref> Invasion of Porphyromonas gingivalis strains into vascular cells and tissue #MMPMID26329158 Olsen I; Progulske-Fox A J Oral Microbiol 2015[]; 7 (ä): ä PMID26329158show ga Porphyromonas gingivalis is considered a major pathogen in adult periodontitis and is also associated with multiple systemic diseases, for example, cardiovascular diseases. One of its most important virulence factors is invasion of host cells. The invasion process includes attachment, entry/internalization, trafficking, persistence, and exit. The present review discusses these processes related to P. gingivalis in cardiovascular cells and tissue. Although most P. gingivalis strains invade, the invasion capacity of strains and the mechanisms of invasion including intracellular trafficking among them differ. This is consistent with the fact that there are significant differences in the pathogenicity of P. gingivalis strains. P. gingivalis invasion mechanisms are also dependent on types of host cells. Although much is known about the invasion process of P. gingivalis, we still have little knowledge of its exit mechanisms. Nevertheless, it is intriguing that P. gingivalis can remain viable in human cardiovascular cells and atherosclerotic plaque and later exit and re-enter previously uninfected host cells. äInvasion of Porphyromonas gingivalis strains into vascular cells and t(epmc).pdf DeepDyve Pubget Overpricing